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Division of Hematology,
Department of Medicine,
Division of Hematology, Department of Medicine
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Depletion of Jak2V617F myeloproliferative neoplasm-propagating stem cells by interferon-α in a murine model of polycythemia vera.
Blood May, 2013 | Pubmed ID: 23487027
RECQL5 Suppresses Oncogenic JAK2-Induced Replication Stress and Genomic Instability.
Cell reports Dec, 2015 | Pubmed ID: 26686625
Mutant Calreticulin Requires Both Its Mutant C-terminus and the Thrombopoietin Receptor for Oncogenic Transformation.
Cancer discovery Apr, 2016 | Pubmed ID: 26951227
Underlying mechanisms of the JAK2V617F mutation in the pathogenesis of myeloproliferative neoplasms.
Der Pathologe Nov, 2016 | Pubmed ID: 27796499
The Development and Use of Janus Kinase 2 Inhibitors for the Treatment of Myeloproliferative Neoplasms.
Hematology/oncology clinics of North America Aug, 2017 | Pubmed ID: 28673391
Kinase Inhibitors in the Treatment of Myeloid Malignancies.
Hematology/oncology clinics of North America Aug, 2017 | Pubmed ID: 28673398
Defining the requirements for the pathogenic interaction between mutant calreticulin and MPL in MPN.
Blood Dec, 2017 | Pubmed ID: 29288169
Brigham and Women's Hospital, Harvard Medical School
Broad Institute
Harvard Medical School
Nouran S. Abdelfattah1,
Ann Mullally1,2,3
1Division of Hematology, Department of Medicine, Brigham and Women's Hospital, Harvard Medical School,
2, Broad Institute,
3Dana-Farber Cancer Institute, Harvard Medical School
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