Janet Richmond

New Data on the Methohexital-Thiopental-Arrhythmia Issue.

Establishing and sculpting the synapse in Drosophila and C. elegans.

The synaptic vesicle cycle: exocytosis and endocytosis in Drosophila and C. elegans.

Defects in synaptic vesicle docking in unc-18 mutants.

Endophilin is required for synaptic vesicle endocytosis by localizing synaptojanin.

The Caenorhabditis elegans unc-63 gene encodes a levamisole-sensitive nicotinic acetylcholine receptor alpha subunit.

Residency training in emergency psychiatry: a model curriculum developed by the education committee of the american association for emergency psychiatry.

SNF-6 is an acetylcholine transporter interacting with the dystrophin complex in Caenorhabditis elegans.

A transmembrane protein required for acetylcholine receptor clustering in Caenorhabditis elegans.

Synaptic vesicle docking: a putative role for the Munc18/Sec1 protein family.

The composition of the GABA receptor at the Caenorhabditis elegans neuromuscular junction.

The Caenorhabditis elegans lev-8 gene encodes a novel type of nicotinic acetylcholine receptor alpha subunit.

acr-16 encodes an essential subunit of the levamisole-resistant nicotinic receptor at the Caenorhabditis elegans neuromuscular junction.

Presynaptic terminals independently regulate synaptic clustering and autophagy of GABAA receptors in Caenorhabditis elegans.

The C2H2 zinc-finger protein SYD-9 is a putative posttranscriptional regulator for synaptic transmission.

UNC-13 and UNC-10/rim localize synaptic vesicles to specific membrane domains.

Tomosyn inhibits synaptic vesicle priming in Caenorhabditis elegans.

Visualization of aggregation of the Rnq1 prion domain and cross-seeding interactions with Sup35NM.

Tomosyn negatively regulates both synaptic transmitter and neuropeptide release at the C. elegans neuromuscular junction.

Regulation of nicotinic receptor trafficking by the transmembrane Golgi protein UNC-50.

Tomosyn negatively regulates CAPS-dependent peptide release at Caenorhabditis elegans synapses.

Synaptic function.

Electrophysiological recordings from the neuromuscular junction of C. elegans.

ITSN-1 controls vesicle recycling at the neuromuscular junction and functions in parallel with DAB-1.

Variant-specific [PSI+] infection is transmitted by Sup35 polymers within [PSI+] aggregates with heterogeneous protein composition.

Direct interactions between C. elegans RAB-3 and Rim provide a mechanism to target vesicles to the presynaptic density.

Eight genes are required for functional reconstitution of the Caenorhabditis elegans levamisole-sensitive acetylcholine receptor.

A secreted complement-control-related protein ensures acetylcholine receptor clustering.

Impaired dense core vesicle maturation in Caenorhabditis elegans mutants lacking Rab2.

UNC-108/RAB-2 and its effector RIC-19 are involved in dense core vesicle maturation in Caenorhabditis elegans.

Regulated lysosomal trafficking as a mechanism for regulating GABAA receptor abundance at synapses in Caenorhabditis elegans.

Neurexin in embryonic Drosophila neuromuscular junctions.

An alpha-catulin homologue controls neuromuscular function through localization of the dystrophin complex and BK channels in Caenorhabditis elegans.

A single immunoglobulin-domain protein required for clustering acetylcholine receptors in C. elegans.

Membrane penetration by synaptotagmin is required for coupling calcium binding to vesicle fusion in vivo.

Differential Regulation of Synaptic Vesicle Tethering and Docking by UNC-18 and TOM-1.

The presynaptic dense projection of the Caenorhabditis elegans cholinergic neuromuscular junction localizes synaptic vesicles at the active zone through SYD-2/liprin and UNC-10/RIM-dependent interactions.

The dystrophin-associated protein complex maintains muscle excitability by regulating Ca(2+)-dependent K(+) (BK) channel localization.

In vivo analysis of conserved C. elegans tomosyn domains.

A transcriptional program promotes remodeling of GABAergic synapses in Caenorhabditis elegans.

Tomosyn-dependent regulation of synaptic transmission is required for a late phase of associative odor memory.

Electrophysiological methods for Caenorhabditis elegans neurobiology.