Ken Nally

Identification of TLR10 as a key mediator of the inflammatory response to Listeria monocytogenes in intestinal epithelial cells and macrophages.

A ghrelin receptor and oxytocin receptor heterocomplex impairs oxytocin mediated signalling.

Human BCL-G regulates secretion of inflammatory chemokines but is dispensable for induction of apoptosis by IFN-γ and TNF-α in intestinal epithelial cells.

A role for the orphan nuclear receptor TLX in the interaction between neural precursor cells and microglia.

Model for Murine Gut Colonization by Bifidobacteria.

Macrophage cytokine responses to commensal Gram-positive Lactobacillus salivarius strains are TLR2-independent and Myd88-dependent.

Exopolysaccharide Blocks Dendritic Cell Maturation and Activation of CD4 T Cells.

Inflammasome Signaling Regulates the Microbial-Neuroimmune Axis and Visceral Pain in Mice.

Regulation of CEACAM Family Members by IBD-Associated Triggers in Intestinal Epithelial Cells, Their Correlation to Inflammation and Relevance to IBD Pathogenesis.

TNF-α synergises with IFN-γ to induce caspase-8-JAK1/2-STAT1-dependent death of intestinal epithelial cells.

Identification of Gut Bacteria such as Lactobacillus johnsonii that Disseminate to Systemic Tissues of Wild Type and MyD88-/- Mice.

Cancer Immunotherapy with Immune Checkpoint Inhibitors-Biomarkers of Response and Toxicity; Current Limitations and Future Promise.

DNA sensor-associated type I interferon signaling is increased in ulcerative colitis and induces JAK-dependent inflammatory cell death in colonic organoids.

Microbial bile salt hydrolase activity influences gene expression profiles and gastrointestinal maturation in infant mice.

An IBD-associated pathobiont synergises with NSAID to promote colitis which is blocked by NLRP3 inflammasome and Caspase-8 inhibitors.

Intracellular glycogen accumulation by human gut commensals as a niche adaptation trait.

Human intestinal organoids: Modeling gastrointestinal physiology and immunopathology - current applications and limitations.