Christian Capo

Survival of Tropheryma whipplei, the agent of Whipple's disease, requires phagosome acidification.

Coxiella burnetii survival in THP-1 monocytes involves the impairment of phagosome maturation: IFN-gamma mediates its restoration and bacterial killing.

IFN-gamma-induced apoptosis and microbicidal activity in monocytes harboring the intracellular bacterium Coxiella burnetii require membrane TNF and homotypic cell adherence.

Bartonella quintana bacteremia and overproduction of interleukin-10: model of bacterial persistence in homeless people.

Dysregulation of cytokines in acute Q fever: role of interleukin-10 and tumor necrosis factor in chronic evolution of Q fever.

Coxiella burnetii avoids macrophage phagocytosis by interfering with spatial distribution of complement receptor 3.

Interleukin-4 induces Coxiella burnetii replication in human monocytes but not in macrophages.

Effect of sex on Coxiella burnetii infection: protective role of 17beta-estradiol.

Lipopolysaccharide from Coxiella burnetii is involved in bacterial phagocytosis, filamentous actin reorganization, and inflammatory responses through Toll-like receptor 4.

Link between impaired maturation of phagosomes and defective Coxiella burnetii killing in patients with chronic Q fever.

Deficient transendothelial migration of leukocytes in Q fever: the role played by interleukin-10.

The two faces of interleukin 10 in human infectious diseases.

Coxiella burnetii stimulates production of RANTES and MCP-1 by mononuclear cells: modulation by adhesion to endothelial cells and its implication in Q fever.

A murine model of infection with Rickettsia prowazekii: implications for pathogenesis of epidemic typhus.

Coxiella burnetii infection in C57BL/6 mice aged 1 or 14 months.

Preeclampsia: impaired inflammatory response mediated by Toll-like receptors.

Infection of endothelial cells with virulent Rickettsia prowazekii increases the transmigration of leukocytes.

Persistent Coxiella burnetii infection in mice overexpressing IL-10: an efficient model for chronic Q fever pathogenesis.

Monocyte deactivation in neutropenic acute respiratory distress syndrome patients treated with granulocyte colony-stimulating factor.

Rickettsial diseases: from Rickettsia-arthropod relationships to pathophysiology and animal models.

The uptake of apoptotic cells drives Coxiella burnetii replication and macrophage polarization: a model for Q fever endocarditis.

Ameobal pathogen mimivirus infects macrophages through phagocytosis.

Epidemic typhus.

New microbicidal functions of tracheal glands: defective anti-infectious response to Pseudomonas aeruginosa in cystic fibrosis.

Intracellular life of Coxiella burnetii in macrophages.

Adipose tissue serves as a reservoir for recrudescent Rickettsia prowazekii infection in a mouse model.

The transcriptional programme of human heart valves reveals the natural history of infective endocarditis.

[Is macrophage polarization the Gordian knot of bacterial infections?].

3D reconstruction of granulomas from transmitted light images implemented for long-time microscope applications.

Sex-related differences in gene expression following Coxiella burnetii infection in mice: potential role of circadian rhythm.

IL-16 promotes T. whipplei replication by inhibiting phagosome conversion and modulating macrophage activation.

Global analysis of circulating immune cells by matrix-assisted laser desorption ionization time-of-flight mass spectrometry.

Coxiella burnetii, the agent of Q fever, replicates within trophoblasts and induces a unique transcriptional response.

Macrophage polarization and bacterial infections.

Orientia tsutsugamushi stimulates an original gene expression program in monocytes: relationship with gene expression in patients with scrub typhus.

The ligands of Numb proteins X1 and X2 are specific markers for chronic Q fever.

Effects of Coxiella burnetii on MAPKinases phosphorylation.

Foxp3(+)CD4(+)CD25(+) regulatory T cells are increased in patients with Coxiella burnetii endocarditis.

The gene expression analysis of blood reveals S100A11 and AQP9 as potential biomarkers of infective endocarditis.

Defective monocyte dynamics in Q fever granuloma deficiency.

Role of innate and adaptive immunity in the control of Q fever.

Whole-cell MALDI-TOF MS: a new tool to assess the multifaceted activation of macrophages.

Overexpression of the Per2 gene in male patients with acute Q fever.

Orientia tsutsugamushi, the causative agent of scrub typhus, induces an inflammatory program in human macrophages.

Impaired stimulation of p38α-MAPK/Vps41-HOPS by LPS from pathogenic Coxiella burnetii prevents trafficking to microbicidal phagolysosomes.

Monocyte responses in the context of Q fever: from a static polarized model to a kinetic model of activation.

Whole-cell MALDI-TOF mass spectrometry: a tool for immune cell analysis and characterization.