Xiangjian Zheng

AngII induces transient phospholipase D activity in the H295R glomerulosa cell model.

Modulation of phospholipase D-mediated phosphatidylglycerol formation by differentiating agents in primary mouse epidermal keratinocytes.

Aquaporin 3 colocates with phospholipase d2 in caveolin-rich membrane microdomains and is downregulated upon keratinocyte differentiation.

Phospholipase d signaling and extracellular signal-regulated kinase-1 and -2 phosphorylation (activation) are required for maximal phorbol ester-induced transglutaminase activity, a marker of keratinocyte differentiation.

Enrichment of regulatory CD4(+)CD25(+) T cells by inhibition of phospholipase D signaling.

A potential role for the phospholipase D2-aquaporin-3 signaling module in early keratinocyte differentiation: production of a phosphatidylglycerol signaling lipid.

Regulation of cardiovascular development and integrity by the heart of glass-cerebral cavernous malformation protein pathway.

Phospholipase D1 is required for angiogenesis of intersegmental blood vessels in zebrafish.

CCM3 signaling through sterile 20-like kinases plays an essential role during zebrafish cardiovascular development and cerebral cavernous malformations.

Aquaporin-3 in keratinocytes and skin: its role and interaction with phospholipase D2.

Dynamic regulation of the cerebral cavernous malformation pathway controls vascular stability and growth.

Cerebral cavernous malformations arise independent of the heart of glass receptor.

The cerebral cavernous malformation pathway controls cardiac development via regulation of endocardial MEKK3 signaling and KLF expression.

Cerebral cavernous malformations arise from endothelial gain of MEKK3-KLF2/4 signalling.

Corrigendum: Cerebral cavernous malformations arise from endothelial gain of MEKK3-KLF2/4 signalling.

Micro-CT Imaging Reveals Mekk3 Heterozygosity Prevents Cerebral Cavernous Malformations in Ccm2-Deficient Mice.

A MicroRNA302-367-Erk1/2-Klf2-S1pr1 Pathway Prevents Tumor Growth via Restricting Angiogenesis and Improving Vascular Stability.

Endothelial TLR4 and the microbiome drive cerebral cavernous malformations.