Arthur T. Suckow

A highly sensitive immunoassay resistant to autoantibody interference for detection of the diabetes-associated autoantigen glutamic acid decarboxylase 65 in blood and other biological samples.

Deletion of JAM-A causes morphological defects in the corneal epithelium.

Release of glutamate decarboxylase-65 into the circulation by injured pancreatic islet beta-cells.

Attenuation of junctional adhesion molecule-A is a contributing factor for breast cancer cell invasion.

Expression of neurexin, neuroligin, and their cytoplasmic binding partners in the pancreatic beta-cells and the involvement of neuroligin in insulin secretion.

An AP-3-dependent mechanism drives synaptic-like microvesicle biogenesis in pancreatic islet beta-cells.

Transcellular neuroligin-2 interactions enhance insulin secretion and are integral to pancreatic β cell function.