Shaun M. Cowley

Histone deacetylase 6 binds polyubiquitin through its zinc finger (PAZ domain) and copurifies with deubiquitinating enzymes.

Genomic binding by the Drosophila Myc, Max, Mad/Mnt transcription factor network.

Functional analysis of the Mad1-mSin3A repressor-corepressor interaction reveals determinants of specificity, affinity, and transcriptional response.

HBP1 and Mad1 repressors bind the Sin3 corepressor PAH2 domain with opposite helical orientations.

The mSin3A chromatin-modifying complex is essential for embryogenesis and T-cell development.

Histone deacetylase 1 (HDAC1), but not HDAC2, controls embryonic stem cell differentiation.

Emphasizing the positive: A role for histone deacetylases in transcriptional activation.

Lysine-specific demethylase 1 regulates the embryonic transcriptome and CoREST stability.

Enhancer decommissioning by LSD1 during embryonic stem cell differentiation.

Histone deacetylase 1 and 2 are essential for normal T-cell development and genomic stability in mice.

The physiological roles of histone deacetylase (HDAC) 1 and 2: complex co-stars with multiple leading parts.

Class I HDACs share a common mechanism of regulation by inositol phosphates.

Histone deacetylase (HDAC) 1 and 2 are essential for accurate cell division and the pluripotency of embryonic stem cells.