Weibin Zhou

Caspy, a zebrafish caspase, activated by ASC oligomerization is required for pharyngeal arch development.

Transmembrane sema4E guides branchiomotor axons to their targets in zebrafish.

accordion, a zebrafish behavioral mutant, has a muscle relaxation defect due to a mutation in the ATPase Ca2+ pump SERCA1.

Zebrafish bandoneon mutants display behavioral defects due to a mutation in the glycine receptor beta-subunit.

Ion-exchange chromatography of hepatitis B virus surface antigen from a recombinant Chinese hamster ovary cell line.

The ROC analysis for different time points during oral glucose tolerance test.

Non-sense mutations in the dihydropyridine receptor beta1 gene, CACNB1, paralyze zebrafish relaxed mutants.

Molecular cloning and expression of two small leucine-rich proteoglycan (SLRP) genes, dspg3l and optcl, in zebrafish.

The -822G/A polymorphism in the promoter region of the MAP4K5 gene is associated with reduced risk of type 2 diabetes in Chinese Hans from Shanghai.

Molecular characterization of recombinant Hepatitis B surface antigen from Chinese hamster ovary and Hansenula polymorpha cells by high-performance size exclusion chromatography and multi-angle laser light scattering.

Assessing 1-h plasma glucose and shape of the glucose curve during oral glucose tolerance test.

Nephronophthisis-associated ciliopathies.

Zebrafish relatively relaxed mutants have a ryanodine receptor defect, show slow swimming and provide a model of multi-minicore disease.

A highly efficient integrated chromatographic procedure for the purification of recombinant hepatitis B surface antigen from Hansenula polymorpha.

The zebrafish ennui behavioral mutation disrupts acetylcholine receptor localization and motor axon stability.

Mutation analysis in nephronophthisis using a combined approach of homozygosity mapping, CEL I endonuclease cleavage, and direct sequencing.

Metabolic characteristics of subjects with normal glucose tolerance and 1-h hyperglycaemia.

Identification and expression of voltage-gated calcium channel beta subunits in Zebrafish.

A systematic approach to mapping recessive disease genes in individuals from outbred populations.

Assaying autophagic activity in transgenic GFP-Lc3 and GFP-Gabarap zebrafish embryos.

Molecular cloning and expression of phospholipase C epsilon 1 in zebrafish.

Integrity of N- and C-termini is important for E. coli Hsp31 chaperone activity.

Individuals with mutations in XPNPEP3, which encodes a mitochondrial protein, develop a nephronophthisis-like nephropathy.

Single-pot biofabrication of zinc sulfide immuno-quantum dots.

Na(v)1.6a is required for normal activation of motor circuits normally excited by tactile stimulation.

Nephrocystin-3 is required for ciliary function in zebrafish embryos.

touché Is required for touch-evoked generator potentials within vertebrate sensory neurons.

The retinitis pigmentosa protein RP2 interacts with polycystin 2 and regulates cilia-mediated vertebrate development.

Characterization of mesonephric development and regeneration using transgenic zebrafish.

Candidate exome capture identifies mutation of SDCCAG8 as the cause of a retinal-renal ciliopathy.

Mutation analysis of 18 nephronophthisis associated ciliopathy disease genes using a DNA pooling and next generation sequencing strategy.

COQ6 mutations in human patients produce nephrotic syndrome with sensorineural deafness.

TRPM7 is required within zebrafish sensory neurons for the activation of touch-evoked escape behaviors.

Aqueous, protein-driven synthesis of transition metal-doped ZnS immuno-quantum dots.

Connexin 39.9 protein is necessary for coordinated activation of slow-twitch muscle and normal behavior in zebrafish.

Biomineralization and size control of stable calcium phosphate core-protein shell nanoparticles: potential for vaccine applications.

Laying out ground rules for protein-aided nanofabrication: ZnO synthesis at 70°C as a case study.

Inducible podocyte injury and proteinuria in transgenic zebrafish.

FAN1 mutations cause karyomegalic interstitial nephritis, linking chronic kidney failure to defective DNA damage repair.

Exome capture reveals ZNF423 and CEP164 mutations, linking renal ciliopathies to DNA damage response signaling.

Mutation of the Mg2+ transporter SLC41A1 results in a nephronophthisis-like phenotype.

Myo1c is an unconventional myosin required for zebrafish glomerular development.

Stac3 is a component of the excitation-contraction coupling machinery and mutated in Native American myopathy.

A system for investigation of biological effects of diagnostic ultrasound on development of zebrafish embryos.

ARHGDIA mutations cause nephrotic syndrome via defective RHO GTPase signaling.

ZMYND10 is mutated in primary ciliary dyskinesia and interacts with LRRC6.

Whole-exome resequencing distinguishes cystic kidney diseases from phenocopies in renal ciliopathies.

ADCK4 mutations promote steroid-resistant nephrotic syndrome through CoQ10 biosynthesis disruption.

Just-in-time vaccines: Biomineralized calcium phosphate core-immunogen shell nanoparticles induce long-lasting CD8(+) T cell responses in mice.

Mutations in EMP2 cause childhood-onset nephrotic syndrome.

Cellular imaging of deep organ using two-photon Bessel light-sheet nonlinear structured illumination microscopy.

Biofabrication of ZnS:Mn luminescent nanocrystals using histidine, hexahistidine, and His-tagged proteins: a comparison study.

Wnt5a Is Necessary for Normal Kidney Development in Zebrafish and Mice.