John R. Grainger

Cooperation between different forms of the human papillomavirus type 1 E4 protein to block cell cycle progression and cellular DNA synthesis.

Helminth immunoregulation: the role of parasite secreted proteins in modulating host immunity.

Microbe-dendritic cell dialog controls regulatory T-cell fate.

Helminth secretions induce de novo T cell Foxp3 expression and regulatory function through the TGF-β pathway.

Generation of pathogenic T(H)17 cells in the absence of TGF-β signalling.

Opposing regulation of the locus encoding IL-17 through direct, reciprocal actions of STAT3 and STAT5.

Essential role for retinoic acid in the promotion of CD4(+) T cell effector responses via retinoic acid receptor alpha.

Proteomic analysis of secretory products from the model gastrointestinal nematode Heligmosomoides polygyrus reveals dominance of venom allergen-like (VAL) proteins.

The role of retinoic acid in tolerance and immunity.

Immune modulation and modulators in Heligmosomoides polygyrus infection.

Heligmosomoides polygyrus elicits a dominant nonprotective antibody response directed against restricted glycan and peptide epitopes.

GATA3 controls Foxp3⁺ regulatory T cell fate during inflammation in mice.

The transcription factors Thpok and LRF are necessary and partly redundant for T helper cell differentiation.

Inflammatory monocytes regulate pathologic responses to commensals during acute gastrointestinal infection.

Retinoic acid controls the homeostasis of pre-cDC-derived splenic and intestinal dendritic cells.

Minimal differentiation of classical monocytes as they survey steady-state tissues and transport antigen to lymph nodes.

Intraluminal containment of commensal outgrowth in the gut during infection-induced dysbiosis.

Thymocyte apoptosis drives the intrathymic generation of regulatory T cells.

Innate and adaptive type 2 immune cell responses in genetically controlled resistance to intestinal helminth infection.

Contextual functions of antigen-presenting cells in the gastrointestinal tract.

Neutrophils worm their way into macrophage long-term memory.