Victoria E. Prince

Knockdown of duplicated zebrafish hoxb1 genes reveals distinct roles in hindbrain patterning and a novel mechanism of duplicate gene retention.

Constructing the hindbrain: insights from the zebrafish.

Zebrafish hox paralogue group 2 genes function redundantly as selector genes to pattern the second pharyngeal arch.

Retinoic acid signaling is required for a critical early step in zebrafish pancreatic development.

Splitting pairs: the diverging fates of duplicated genes.

Hox gene misexpression and cell-specific lesions reveal functionality of homeotically transformed neurons.

Zebrafish gcm2 is required for gill filament budding from pharyngeal ectoderm.

Retinoids signal directly to zebrafish endoderm to specify insulin-expressing beta-cells.

Origin of the zebrafish endocrine and exocrine pancreas.

A new time-scale for ray-finned fish evolution.

Repression of the hindbrain developmental program by Cdx factors is required for the specification of the vertebrate spinal cord.

Zebrafish Hoxb1a regulates multiple downstream genes including prickle1b.

Consequences of hoxb1 duplication in teleost fish.

Zic1 and Zic4 regulate zebrafish roof plate specification and hindbrain ventricle morphogenesis.

Cdx4 is required in the endoderm to localize the pancreas and limit beta-cell number.

Current perspectives in zebrafish reverse genetics: moving forward.

On the diabetic menu: zebrafish as a model for pancreas development and function.

Cyp26 enzymes function in endoderm to regulate pancreatic field size.

The autism susceptibility gene met regulates zebrafish cerebellar development and facial motor neuron migration.

Model organisms inform the search for the genes and developmental pathology underlying malformations of the human hindbrain.

Multiple mechanisms mediate motor neuron migration in the zebrafish hindbrain.

In vitro processing and secretion of mutant insulin proteins that cause permanent neonatal diabetes.

Recent advances in pancreas development: from embryonic pathways to programming renewable sources of beta cells.

Prickle1b mediates interpretation of migratory cues during zebrafish facial branchiomotor neuron migration.

Blood sugar measurement in zebrafish reveals dynamics of glucose homeostasis.

Zebrafish Prickle1b mediates facial branchiomotor neuron migration via a farnesylation-dependent nuclear activity.

Zebrafish mnx1 controls cell fate choice in the developing endocrine pancreas.