Anthony J. Baker

Alpha(1)-adrenoceptor subtypes mediate negative inotropy in myocardium from alpha(1A/C)-knockout and wild type mice.

Alpha 1-adrenergic receptor responses in alpha 1AB-AR knockout mouse hearts suggest the presence of alpha 1D-AR.

Abnormal myocardial contraction in alpha(1A)- and alpha(1B)-adrenoceptor double-knockout mice.

Cardiac transgenesis with the tetracycline transactivator changes myocardial function and gene expression.

Cardiac transgenic matrix metalloproteinase-2 expression directly induces impaired contractility.

Sustained preconditioning induced by cardiac transgenesis with the tetracycline transactivator.

Alpha1-adrenergic receptors prevent a maladaptive cardiac response to pressure overload.

Contrasting inotropic responses to alpha1-adrenergic receptor stimulation in left versus right ventricular myocardium.

Transgenic MMP-2 expression induces latent cardiac mitochondrial dysfunction.

Expression of a Gi-coupled receptor in the heart causes impaired Ca2+ handling, myofilament injury, and dilated cardiomyopathy.

A computationally efficient formal optimization of regional myocardial contractility in a sheep with left ventricular aneurysm.

Heart failure switches the RV alpha1-adrenergic inotropic response from negative to positive.

Distinctive ERK and p38 signaling in remote and infarcted myocardium during post-MI remodeling in the mouse.

Refueling the heart: Using 2-deoxy-ATP to enhance cardiac contractility.