Chunsik Lee

A fragment of histidine-rich glycoprotein is a potent inhibitor of tumor vascularization.

Transgenic expression of dominant negative tuberin through a strong constitutive promoter results in a tissue-specific tuberous sclerosis phenotype in the skin and brain.

Minimal active domain and mechanism of action of the angiogenesis inhibitor histidine-rich glycoprotein.

Signal transduction in endothelial cells by the angiogenesis inhibitor histidine-rich glycoprotein targets focal adhesions.

Type 3 cystatins; fetuins, kininogen and histidine-rich glycoprotein.

VEGF-B is dispensable for blood vessel growth but critical for their survival, and VEGF-B targeting inhibits pathological angiogenesis.

VEGF-B: a survival, or an angiogenic factor?

Platelet-derived growth factor-DD targeting arrests pathological angiogenesis by modulating glycogen synthase kinase-3beta phosphorylation.

Survival effect of PDGF-CC rescues neurons from apoptosis in both brain and retina by regulating GSK3beta phosphorylation.

PDGF-CC blockade inhibits pathological angiogenesis by acting on multiple cellular and molecular targets.

VEGF-independent angiogenic pathways induced by PDGF-C.

Complicated life, complicated VEGF-B.