Jeffrey H. Withey

Flexibility of Vibrio cholerae ToxT in transcription activation of genes having altered promoter spacing.

Bicarbonate Induces Vibrio cholerae virulence gene expression by enhancing ToxT activity.

Genetic screening for bacterial mutants in liquid growth media by fluorescence-activated cell sorting.

Termination of Vibrio cholerae virulence gene expression is mediated by proteolysis of the major virulence activator, ToxT.

Identification and characterization of the functional toxboxes in the Vibrio cholerae cholera toxin promoter.

Zebrafish as a natural host model for Vibrio cholerae colonization and transmission.

Zebrafish as a model for zoonotic aquatic pathogens.

Bicarbonate increases binding affinity of Vibrio cholerae ToxT to virulence gene promoters.

A small unstructured region in Vibrio cholerae ToxT mediates the response to positive and negative effectors and ToxT proteolysis.

A metalloprotease secreted by the type II secretion system links Vibrio cholerae with collagen.

Mechanism for inhibition of Vibrio cholerae ToxT activity by the unsaturated fatty acid components of bile.

Conjugated Linoleic Acid Reduces Cholera Toxin Production In Vitro and In Vivo by Inhibiting Vibrio cholerae ToxT Activity.

Zebrafish as a novel model for non-typhoidal Salmonella pathogenesis, transmission and vaccine efficacy.

Quantifying enterotoxicity in a zebrafish infection model.

[C] Linoleic Acid Uptake and Fractionation Assay in .