Elena Galkina

Interactions of C-Reactive Protein and Serum Amyloid P Component with Interleukin-8 and Their Role in Regulation of Neutrophil Functions.

L-selectin shedding does not regulate constitutive T cell trafficking but controls the migration pathways of antigen-activated T lymphocytes.

Autoperfused mouse flow chamber reveals synergistic neutrophil accumulation through P-selectin and E-selectin.

GRO family chemokines are specialized for monocyte arrest from flow.

Sequential recruitment of neutrophils into lung and bronchoalveolar lavage fluid in LPS-induced acute lung injury.

Preferential migration of effector CD8+ T cells into the interstitium of the normal lung.

Leukocyte recruitment and vascular injury in diabetic nephropathy.

Double jeopardy: how soluble P-selectin activates leukocytes in peripheral arterial occlusive disease.

Lymphocyte recruitment into the aortic wall before and during development of atherosclerosis is partially L-selectin dependent.

T lymphocyte rolling and recruitment into peripheral lymph nodes is regulated by a saturable density of L-selectin (CD62L).

Vascular adhesion molecules in atherosclerosis.

CXCR6 promotes atherosclerosis by supporting T-cell homing, interferon-gamma production, and macrophage accumulation in the aortic wall.

Leukocyte influx in atherosclerosis.

CD40 ligand promotes Mac-1 expression, leukocyte recruitment, and neointima formation after vascular injury.

Mechanisms by which diabetes increases cardiovascular disease.

Immune and inflammatory mechanisms of atherosclerosis (*).

Induction of CCR2-dependent macrophage accumulation by oxidized phospholipids in the air-pouch model of inflammation.

Blockade of interleukin-17A results in reduced atherosclerosis in apolipoprotein E-deficient mice.

B-cell aortic homing and atheroprotection depend on Id3.

Accelerated atherosclerosis in apoe-/- mice heterozygous for the insulin receptor and the insulin receptor substrate-1.

The IL-17A/IL-17RA Axis Plays a Proatherogenic Role via the Regulation of Aortic Myeloid Cell Recruitment.