Ranjan Dutta

Dysmyelinated lower motor neurons retract and regenerate dysfunctional synaptic terminals.

Mitochondrial dysfunction as a cause of axonal degeneration in multiple sclerosis patients.

[Pathology and definition of multiple sclerosis].

Pathogenesis of axonal and neuronal damage in multiple sclerosis.

Activation of the ciliary neurotrophic factor (CNTF) signalling pathway in cortical neurons of multiple sclerosis patients.

Mechanisms of neuronal dysfunction and degeneration in multiple sclerosis.

Gene expression profiling in multiple sclerosis brain.

Demyelination causes synaptic alterations in hippocampi from multiple sclerosis patients.

Lipopolysaccharide-induced microglial activation and neuroprotection against experimental brain injury is independent of hematogenous TLR4.

Cortical remyelination: a new target for repair therapies in multiple sclerosis.

Hippocampal demyelination and memory dysfunction are associated with increased levels of the neuronal microRNA miR-124 and reduced AMPA receptors.

Activated microglia enhance neurogenesis via trypsinogen secretion.

Gene expression changes underlying cortical pathology: clues to understanding neurological disability in multiple sclerosis.

Epigenome-wide differences in pathology-free regions of multiple sclerosis-affected brains.

Axonal loss in multiple sclerosis: causes and mechanisms.

Relapsing and progressive forms of multiple sclerosis: insights from pathology.

Microglial displacement of inhibitory synapses provides neuroprotection in the adult brain.

Promoting remyelination in multiple sclerosis: current drugs and future prospects.

A mouse model for testing remyelinating therapies.

Alterations in CA1 hippocampal synapses in a mouse model of fragile X syndrome.

Much, if not all, of the cortical damage in MS can be attributed to the microglial cell - No.

Distinct Gene Profiles of Bone Marrow-Derived Macrophages and Microglia During Neurotropic Coronavirus-Induced Demyelination.

CK2 inhibition confers functional protection to young and aging axons against ischemia by differentially regulating the CDK5 and AKT signaling pathways.

Current advancements in promoting remyelination in multiple sclerosis.

Constitutional mislocalization of Pten drives precocious maturation in oligodendrocytes and aberrant myelination in model of autism spectrum disorder.

Proteomic Approaches to Decipher Mechanisms Underlying Pathogenesis in Multiple Sclerosis Patients.

Expression of disease-related miRNAs in white-matter lesions of progressive multiple sclerosis brains.

pHERV-W envelope protein fuels microglial cell-dependent damage of myelinated axons in multiple sclerosis.