Khalid Azizi

ATF6 Decreases Myocardial Ischemia/Reperfusion Damage and Links ER Stress and Oxidative Stress Signaling Pathways in the Heart.

Delayed Management of Partial Aortic Valve Avulsion After Transcatheter Aortic Valve Replacement.

Pharmacologic ATF6 activation confers global protection in widespread disease models by reprograming cellular proteostasis.

The ER Unfolded Protein Response Effector, ATF6, Reduces Cardiac Fibrosis and Decreases Activation of Cardiac Fibroblasts.

Proteomic analysis of the cardiac myocyte secretome reveals extracellular protective functions for the ER stress response.