Henrik Hagberg

Function and Biomarkers of the Blood-Brain Barrier in a Neonatal Germinal Matrix Haemorrhage Model.

[Spontaneous premature birth: physiopathology, predictors and management. The frequency is constant--early detection can improve therapeutic possibilities].

Antenatal brain injury: aetiology and possibilities of prevention.

Models of white matter injury: comparison of infectious, hypoxic-ischemic, and excitotoxic insults.

Sequelae of chorioamnionitis.

[Increased mortality among infants born at night. A reflection of inadequate perinatal intensive care resources in the night-time?].

No correlation between cerebral palsy and cytokines in postnatal blood of preterms.

PARP-1 gene disruption in mice preferentially protects males from perinatal brain injury.

Mitochondrial impairment in the developing brain after hypoxia-ischemia.

[Chorioamnionitis--risk factor of cerebral palsy in fullterm infants].

[A new monitoring method for safer child delivery. Lower number of metabolic acidosis cases with fetal ECG and cardiotocography].

Preconditioning and the developing brain.

Effect of inflammation on central nervous system development and vulnerability.

Brain injury in preterm infants--what can the obstetrician do?

Developmental shift of cyclophilin D contribution to hypoxic-ischemic brain injury.

White matter damage after chronic subclinical inflammation in newborn mice.

Nuclear translocation and calpain-dependent reduction of Bcl-2 after neonatal cerebral hypoxia-ischemia.

Lipopolysaccharide sensitizes neonatal hypoxic-ischemic brain injury in a MyD88-dependent manner.

The role of Toll-like receptors in perinatal brain injury.

Galectin-3 contributes to neonatal hypoxic-ischemic brain injury.

Attenuation of reactive gliosis does not affect infarct volume in neonatal hypoxic-ischemic brain injury in mice.

Neuroprotective effect of Bax-inhibiting peptide on neonatal brain injury.

Systemic stimulation of TLR2 impairs neonatal mouse brain development.

Genetic inhibition of caspase-2 reduces hypoxic-ischemic and excitotoxic neonatal brain injury.

Safety aspects of longitudinal administration of IGF-I/IGFBP-3 complex in neonatal mice.

Trace fear conditioning detects hypoxic-ischemic brain injury in neonatal mice.

Isolation of brain mitochondria from neonatal mice.

Delayed cortical impairment following lipopolysaccharide exposure in preterm fetal sheep.

Inflammatory-induced hibernation in the fetus: priming of fetal sheep metabolism correlates with developmental brain injury.

Combined effect of hypothermia and caspase-2 gene deficiency on neonatal hypoxic-ischemic brain injury.

Inflammation during fetal and neonatal life: implications for neurologic and neuropsychiatric disease in children and adults.

Molecular mechanisms of neonatal brain injury.

Characterization of phenotype markers and neuronotoxic potential of polarised primary microglia in vitro.

Time-dependent effects of systemic lipopolysaccharide injection on regulators of antioxidant defence Nrf2 and PGC-1α in the neonatal rat brain.

Receptor for complement peptide C3a: a therapeutic target for neonatal hypoxic-ischemic brain injury.

Innate defense regulator peptide 1018 protects against perinatal brain injury.

Tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) signaling and cell death in the immature central nervous system after hypoxia-ischemia and inflammation.

Microglia toxicity in preterm brain injury.

Targeting the vasculature for cerebroprotection in the immature brain.

The immune response after hypoxia-ischemia in a mouse model of preterm brain injury.

Livebirth after uterus transplantation.

The effect of osteopontin and osteopontin-derived peptides on preterm brain injury.

Brain barrier properties and cerebral blood flow in neonatal mice exposed to cerebral hypoxia-ischemia.

The role of inflammation in perinatal brain injury.

Transvaginal sonographic evaluation of cervical length in the second trimester of asymptomatic singleton pregnancies, and the risk of preterm delivery.

Does Caspase-6 Have a Role in Perinatal Brain Injury?

Staphylococcus epidermidis Bacteremia Induces Brain Injury in Neonatal Mice via Toll-like Receptor 2-Dependent and -Independent Pathways.

Specific Lipopolysaccharide Serotypes Induce Differential Maternal and Neonatal Inflammatory Responses in a Murine Model of Preterm Labor.

Mitochondrial Optic Atrophy (OPA) 1 Processing Is Altered in Response to Neonatal Hypoxic-Ischemic Brain Injury.

Perinatal brain damage: The term infant.

Heme Oxygenase-1 Mediates Neuroprotection Conferred by Argon in Combination with Hypothermia in Neonatal Hypoxia-Ischemia Brain Injury.

One uterus bridging three generations: first live birth after mother-to-daughter uterus transplantation.

Modeling hormonal and inflammatory contributions to preterm and term labor using uterine temporal transcriptomics.

TLR2-mediated leukocyte trafficking to the developing brain.

Immune responses in perinatal brain injury.

Temporal Characterization of Microglia/Macrophage Phenotypes in a Mouse Model of Neonatal Hypoxic-Ischemic Brain Injury.

Intranasal C3a treatment ameliorates cognitive impairment in a mouse model of neonatal hypoxic-ischemic brain injury.

Effect of Trp53 gene deficiency on brain injury after neonatal hypoxia-ischemia.

Systemic activation of Toll-like receptor 2 suppresses mitochondrial respiration and exacerbates hypoxic-ischemic injury in the developing brain.

Implicating Receptor Activator of NF-κB (RANK)/RANK Ligand Signalling in Microglial Responses to Toll-Like Receptor Stimuli.

Increase of neuronal injury markers Tau and neurofilament light proteins in umbilical blood after intrapartum asphyxia.

Mitochondria, Bioenergetics and Excitotoxicity: New Therapeutic Targets in Perinatal Brain Injury.

Chorioamnionitis in the Development of Cerebral Palsy: A Meta-analysis and Systematic Review.

Cell Death in the Developing Brain after Hypoxia-Ischemia.

Oxidative stress and endoplasmic reticulum (ER) stress in the development of neonatal hypoxic-ischaemic brain injury.

γδ T Cells Contribute to Injury in the Developing Brain.

γδT cells but not αβT cells contribute to sepsis-induced white matter injury and motor abnormalities in mice.

Mitochondrial dynamics, mitophagy and biogenesis in neonatal hypoxic-ischaemic brain injury.

Lymphocytes Contribute to the Pathophysiology of Neonatal Brain Injury.

TWEAK Receptor Deficiency Has Opposite Effects on Female and Male Mice Subjected to Neonatal Hypoxia-Ischemia.

Blood-based cerebral biomarkers in preeclampsia: Plasma concentrations of NfL, tau, S100B and NSE during pregnancy in women who later develop preeclampsia - A nested case control study.

Neuroprotective exendin-4 enhances hypothermia therapy in a model of hypoxic-ischaemic encephalopathy.

Myelination induction by a histamine H3 receptor antagonist in a mouse model of preterm white matter injury.

Peripheral myeloid cells contribute to brain injury in male neonatal mice.

Lack of the brain-specific isoform of apoptosis-inducing factor aggravates cerebral damage in a model of neonatal hypoxia-ischemia.

Lipopolysaccharide-induced alteration of mitochondrial morphology induces a metabolic shift in microglia modulating the inflammatory response in vitro and in vivo.

Choroid plexus transcriptome and ultrastructure analysis reveals a TLR2-specific chemotaxis signature and cytoskeleton remodeling in leukocyte trafficking.

Long-term Risk of Neuropsychiatric Disease After Exposure to Infection In Utero.

The Role of Mitochondrial and Endoplasmic Reticulum Reactive Oxygen Species Production in Models of Perinatal Brain Injury.

Dysmaturation of Somatostatin Interneurons Following Umbilical Cord Occlusion in Preterm Fetal Sheep.

Positive and negative conditioning in the neonatal brain.

Post-mortem Characterisation of a Case With an Variant, Agenesis of the Corpus Callosum and Neuronal Heterotopia.

Microbial invasion of the amniotic cavity is associated with impaired cognitive and motor function at school age in preterm children.

N-acetylcysteine inhibits bacterial lipopeptide-mediated neutrophil transmigration through the choroid plexus in the developing brain.

Overexpression of apoptosis inducing factor aggravates hypoxic-ischemic brain injury in neonatal mice.

Inhibiting the interaction between apoptosis-inducing factor and cyclophilin A prevents brain injury in neonatal mice after hypoxia-ischemia.

Severe maternal morbidity and mortality associated with COVID-19: The risk should not be downplayed.

Neuroprotective Effects of Diabetes Drugs for the Treatment of Neonatal Hypoxia-Ischemia Encephalopathy.

Type 2 Innate Lymphoid Cells Accumulate in the Brain After Hypoxia-Ischemia but Do Not Contribute to the Development of Preterm Brain Injury.

Induction of labour at 41 weeks or expectant management until 42 weeks: A systematic review and an individual participant data meta-analysis of randomised trials.

Neuroprotection offered by mesenchymal stem cells in perinatal brain injury: Role of mitochondria, inflammation, and reactive oxygen species.

Efficacy and safety of oral misoprostol vs transvaginal balloon catheter for labor induction: An observational study within the SWEdish Postterm Induction Study (SWEPIS).

Effect of second-trimester sonographic cervical length on the risk of spontaneous preterm delivery in different risk groups: A prospective observational multicenter study.

Therapies for neonatal encephalopathy: Targeting the latent, secondary and tertiary phases of evolving brain injury.

Single-cell atlas reveals meningeal leukocyte heterogeneity in the developing mouse brain.

Induction of labour at 41 weeks of gestation versus expectant management and induction of labour at 42 weeks of gestation: A cost-effectiveness analysis.

White matter injury but not germinal matrix hemorrhage induces elevated osteopontin expression in human preterm brains.

New possibilities for neuroprotection in neonatal hypoxic-ischemic encephalopathy.

N-Acetyl Cysteine Restores Sirtuin-6 and Decreases HMGB1 Release Following Lipopolysaccharide-Sensitized Hypoxic-Ischemic Brain Injury in Neonatal Mice.

C3a Receptor Signaling Inhibits Neurodegeneration Induced by Neonatal Hypoxic-Ischemic Brain Injury.

Induction of Mitochondrial Fragmentation and Mitophagy after Neonatal Hypoxia-Ischemia.

Temporal brain transcriptome analysis reveals key pathological events after germinal matrix hemorrhage in neonatal rats.

Maternal and fetal serum concentrations of magnesium after administration of a 6-g bolus dose of magnesium sulfate (MgSO ) to women with imminent preterm delivery.

Reproductive, obstetric, and long-term health outcome after uterus transplantation: results of the first clinical trial.

Microbial and human transcriptome in vaginal fluid at midgestation: Association with spontaneous preterm delivery.

Two different isoforms of osteopontin modulate myelination and axonal integrity.

Mechanisms of Tertiary Neurodegeneration after Neonatal Hypoxic-Ischemic Brain Damage.

A follow up on the feasibility after national implementation of magnesium sulfate for neuroprotection prior to preterm birth.

Neuro-Specific and Immuno-Inflammatory Biomarkers in Umbilical Cord Blood in Neonatal Hypoxic-Ischemic Encephalopathy.

Therapeutic Effect of Nicotinamide Mononucleotide for Hypoxic-Ischemic Brain Injury in Neonatal Mice.