Cecilia Flores

Chronic phencyclidine treatment increases dendritic spine density in prefrontal cortex and nucleus accumbens neurons.

Netrin-1 receptor-deficient mice show enhanced mesocortical dopamine transmission and blunted behavioural responses to amphetamine.

Altered netrin-1 receptor expression in dopamine terminal regions following neonatal ventral hippocampal lesions in the rat.

The 31st annual meeting of the Canadian College of Neuropsychopharmacology: Toronto, Ontario, June 6-9, 2008.

Alcohol dependence-related increase of glial cell density in the anterior cingulate cortex of suicide completers.

Post-pubertal emergence of a dopamine phenotype in netrin-1 receptor-deficient mice.

Netrin-1 receptor in the ventral tegmental area is required for sensitization to amphetamine.

Prenatal inflammation-induced hypoferremia alters dopamine function in the adult offspring in rat: relevance for schizophrenia.

Peri-pubertal emergence of UNC-5 homologue expression by dopamine neurons in rodents.

Role of netrin-1 in the organization and function of the mesocorticolimbic dopamine system.

Abolition of the behavioral phenotype of adult netrin-1 receptor deficient mice by exposure to amphetamine during the juvenile period.

The netrin receptor DCC is required in the pubertal organization of mesocortical dopamine circuitry.

Leptin and interleukin-6 alter the function of mesolimbic dopamine neurons in a rodent model of prenatal inflammation.

Juvenile exposure to methylphenidate reduces cocaine reward and alters netrin-1 receptor expression in adulthood.

Association between schizophrenia and genetic variation in DCC: a case-control study.

Adolescence: a time of transition for the phenotype of dcc heterozygous mice.

unc5c haploinsufficient phenotype: striking similarities with the dcc haploinsufficiency model.

Haloperidol treatment downregulates DCC expression in the ventral tegmental area.

The Netrin-1 receptor DCC is a regulator of maladaptive responses to chronic morphine administration.

Amphetamine in adolescence disrupts the development of medial prefrontal cortex dopamine connectivity in a DCC-dependent manner.

dcc Haploinsufficiency results in blunted sensitivity to cocaine enhancement of reward seeking.

Resilience to amphetamine in mouse models of netrin-1 haploinsufficiency: role of mesocortical dopamine.

Mesocortical Dopamine Phenotypes in Mice Lacking the Sonic Hedgehog Receptor Cdon.

DCC Confers Susceptibility to Depression-like Behaviors in Humans and Mice and Is Regulated by miR-218.

Dcc haploinsufficiency regulates dopamine receptor expression across postnatal lifespan.

A non-invasive restraining system for awake mouse imaging.

DCC Receptors Drive Prefrontal Cortex Maturation by Determining Dopamine Axon Targeting in Adolescence.

Making Dopamine Connections in Adolescence.

Adolescence and Reward: Making Sense of Neural and Behavioral Changes Amid the Chaos.

Non-Contingent Exposure to Amphetamine in Adolescence Recruits miR-218 to Regulate Dcc Expression in the VTA.

An optimized immunohistochemistry protocol for detecting the guidance cue Netrin-1 in neural tissue.

Dopamine Development in the Mouse Orbital Prefrontal Cortex Is Protracted and Sensitive to Amphetamine in Adolescence.

Mesocorticolimbic Connectivity and Volumetric Alterations in Mutation Carriers.

Guidance cues: linking drug use in adolescence with psychiatric disorders.

Early Adolescence is a Critical Period for the Maturation of Inhibitory Behavior.

Neural function in DCC mutation carriers with and without mirror movements.

MiR-218: a molecular switch and potential biomarker of susceptibility to stress.

DCC-related developmental effects of abused- versus therapeutic-like amphetamine doses in adolescence.

The Netrin-1/DCC guidance system: dopamine pathway maturation and psychiatric disorders emerging in adolescence.

Low-cost conditioned place preference setup including video recording and analysis of behaviour.

The Netrin-1/DCC Guidance Cue Pathway as a Molecular Target in Depression: Translational Evidence.

Dopamine Axon Targeting in the Nucleus Accumbens in Adolescence Requires Netrin-1.

gene network in the prefrontal cortex is associated with total brain volume in childhood.

miR-218 in Adolescence Predicts and Mediates Vulnerability to Stress.

Unique effects of social defeat stress in adolescent male mice on the Netrin-1/DCC pathway, prefrontal cortex dopamine and cognition (Social stress in adolescent vs. adult male mice).

MicroRNA regulation of prefrontal cortex development and psychiatric risk in adolescence.

MicroRNAs as promising peripheral sensors of prefrontal cortex developmental trajectory and psychiatric risk.

Mechanisms of cortical development: From the embryo to adulthood.

Mesocorticolimbic Dopamine Pathways Across Adolescence: Diversity in Development.

Custom-Built Operant Conditioning Setup for Calcium Imaging and Cognitive Testing in Freely Moving Mice.

Corticolimbic DCC gene co-expression networks as predictors of impulsivity in children.

Reduced dopamine release in Dcc haploinsufficiency male mice abolishes the rewarding effects of cocaine but not those of morphine and ethanol.

We're not in Kansas anymore: ectopic dopaminergic terminals as an explanation for the positive symptoms in psychiatric pathology.

Awakening the dormant: Role of axonal guidance cues in stress-induced reorganization of the adult prefrontal cortex leading to depression-like behavior.

Gains and Losses: Resilience to Social Defeat Stress in Adolescent Female Mice.

Amphetamine disrupts dopamine axon growth in adolescence by a sex-specific mechanism in mice.

Relationship between insulin and Netrin-1/DCC guidance cue pathway regulation in the prefrontal cortex of rodents exposed to prenatal dietary restriction.

Novel Functional Genomics Approaches Bridging Neuroscience and Psychiatry.

Axo-glial interactions between midbrain dopamine neurons and oligodendrocyte lineage cells in the anterior corpus callosum.

Preparation and processing of dried blood spots for microRNA sequencing.

RGMa and Neogenin control dendritic spine morphogenesis via WAVE Regulatory Complex-mediated actin remodeling.

Rewiring the future: drugs abused in adolescence may predispose to mental illness in adult life by altering dopamine axon growth.