Gianrico Farrugia

Sodium current in human jejunal circular smooth muscle cells.

Paraneoplastic dysmotility: loss of interstitial cells of Cajal.

alpha(1C) (Ca(V)1.2) L-type calcium channel mediates mechanosensitive calcium regulation.

The Kv2.2 alpha subunit contributes to delayed rectifier K(+) currents in myocytes from rabbit corpus cavernosum.

Syntrophin gamma 2 regulates SCN5A gating by a PDZ domain-mediated interaction.

Cytoskeletal modulation of sodium current in human jejunal circular smooth muscle cells.

Chronic intestinal pseudoobstruction associated with altered interstitial cells of cajal networks.

Cholinergic denervation in diverticular disease.

A major role for carbon monoxide as an endogenous hyperpolarizing factor in the gastrointestinal tract.

Sodium current in human intestinal interstitial cells of Cajal.

Ion channels in gastrointestinal smooth muscle and interstitial cells of Cajal.

Heme: a determinant of life and death in renal tubular epithelial cells.

The role of carbon monoxide in the gastrointestinal tract.

Nitric oxide in gastrointestinal health and disease.

Carbon monoxide activates human intestinal smooth muscle L-type Ca2+ channels through a nitric oxide-dependent mechanism.

Meal-induced dysphagia and otalgia secondary to a pyriform sinus fistula.

Intercellular coupling of interstitial cells of cajal in the digestive tract.

Effect of mibefradil on sodium and calcium currents.

Effect of modulation of serotonergic, cholinergic, and nitrergic pathways on murine fundic size and compliance measured by ultrasonomicrometry.

Defects in cGMP-PKG pathway contribute to impaired NO-dependent responses in hepatic stellate cells upon activation.

Deciphering the molecular basis for the pacemaker signal.

Gastrointestinal symptoms in families of patients with an SCN5A-encoded cardiac channelopathy: evidence of an intestinal channelopathy.

Serotonergic modulation of murine fundic tone.

Ca2+ handling in human interstitial cells of Cajal.

Membrane potential gradient is carbon monoxide-dependent in mouse and human small intestine.

Mechanisms of disease: the pathological basis of gastroparesis--a review of experimental and clinical studies.

Exogenous serotonin regulates proliferation of interstitial cells of Cajal in mouse jejunum through 5-HT2B receptors.

Determination of gastric emptying in nonobese diabetic mice.

Evolving concepts in the cellular control of gastrointestinal motility: neurogastroenterology and enteric sciences.

Evaluation of endoscopic approaches for deep gastric-muscle-wall biopsies: what works?

Endoscopic "no hole" full-thickness biopsy of the stomach to detect myenteric ganglia.

A mutation in telethonin alters Nav1.5 function.

Enteric autoantibodies and gut motility disorders.

Production of the gaseous signal molecule hydrogen sulfide in mouse tissues.

Changes in the gastric enteric nervous system and muscle: a case report on two patients with diabetic gastroparesis.

Syntrophin mutation associated with long QT syndrome through activation of the nNOS-SCN5A macromolecular complex.

Heme oxygenase-1 protects interstitial cells of Cajal from oxidative stress and reverses diabetic gastroparesis.

Sodium channel mutation in irritable bowel syndrome: evidence for an ion channelopathy.

Lysophosphatidyl choline modulates mechanosensitive L-type Ca2+ current in circular smooth muscle cells from human jejunum.

Diagnostic challenges of motility disorders: optimal detection of CD117+ interstitial cells of Cajal.

Gastrointestinal neuromuscular pathology: guidelines for histological techniques and reporting on behalf of the Gastro 2009 International Working Group.

Ano1 is a selective marker of interstitial cells of Cajal in the human and mouse gastrointestinal tract.

The alpha1H Ca2+ channel subunit is expressed in mouse jejunal interstitial cells of Cajal and myocytes.

Neural autoantibody profile of primary achalasia.

Protein kinase C{gamma} mediates regulation of proliferation by the serotonin 5-hydroxytryptamine receptor 2B.

Endogenous production of H2S in the gastrointestinal tract: still in search of a physiologic function.

Physiology, injury, and recovery of interstitial cells of Cajal: basic and clinical science.

The Mayo Dysphagia Questionnaire-30: documentation of reliability and validity of a tool for interventional trials in adults with esophageal disease.

CD206-positive M2 macrophages that express heme oxygenase-1 protect against diabetic gastroparesis in mice.

T-type Ca(2+) channel modulation by otilonium bromide.

Carbon monoxide reverses diabetic gastroparesis in NOD mice.

Tissue-specific mathematical models of slow wave entrainment in wild-type and 5-HT(2B) knockout mice with altered interstitial cells of Cajal networks.

Loss of Kitlow progenitors, reduced stem cell factor and high oxidative stress underlie gastric dysfunction in progeric mice.

The London Classification of gastrointestinal neuromuscular pathology: report on behalf of the Gastro 2009 International Working Group.

Psychological dysfunction is associated with symptom severity but not disease etiology or degree of gastric retention in patients with gastroparesis.

Kitlow stem cells cause resistance to Kit/platelet-derived growth factor alpha inhibitors in murine gastrointestinal stromal tumors.

Diabetic gastroparesis: what we have learned and had to unlearn in the past 5 years.

Epidemiology, mechanisms, and management of diabetic gastroparesis.

Clinical features of idiopathic gastroparesis vary with sex, body mass, symptom onset, delay in gastric emptying, and gastroparesis severity.

Mechanosensitivity of Nav1.5, a voltage-sensitive sodium channel.

Esophageal papillomas and skin abnormalities. Focal dermal hypoplasia (Goltz syndrome) manifesting with esophageal papillomatosis.

Cellular changes in diabetic and idiopathic gastroparesis.

Altered expression of Ano1 variants in human diabetic gastroparesis.

Gastrointestinal neuromuscular pathology in chronic constipation.

Hydrogen sulfide is a partially redox-independent activator of the human jejunum Na+ channel, Nav1.5.

Characteristics of patients with chronic unexplained nausea and vomiting and normal gastric emptying.

Bloating in gastroparesis: severity, impact, and associated factors.

Reply.

Hydrogen sulfide modulates contractile function in rat jejunum.

Loss of NRF2 impairs gastric nitrergic stimulation and function.

Dietary intake and nutritional deficiencies in patients with diabetic or idiopathic gastroparesis.

Biophysically based modeling of the interstitial cells of cajal: current status and future perspectives.

A stochastic multi-scale model of electrical function in normal and depleted ICC networks.

Similarities and differences between diabetic and idiopathic gastroparesis.

Ultrastructural differences between diabetic and idiopathic gastroparesis.

Ano1 as a regulator of proliferation.

Quantification of gastrointestinal sodium channelopathy.

Inhaled carbon monoxide attenuates myocardial inflammatory cytokine expression in a rat model of cardiopulmonary bypass.

Targeting ion channels for the treatment of gastrointestinal motility disorders.

Ranolazine decreases mechanosensitivity of the voltage-gated sodium ion channel Na(v)1.5: a novel mechanism of drug action.

The places to be for neurogastroenterologists.

Abnormal initiation and conduction of slow-wave activity in gastroparesis, defined by high-resolution electrical mapping.

Membrane permeable local anesthetics modulate Na(V)1.5 mechanosensitivity.

Toward biphasic computed tomography (CT) enteric contrast: material classification of luminal bismuth and mural iodine in a small-bowel phantom using dual-energy CT.

Inhibition of cell proliferation by a selective inhibitor of the Ca(2+)-activated Cl(-) channel, Ano1.

Ranolazine inhibits shear sensitivity of endogenous Na+ current and spontaneous action potentials in HL-1 cells.

Endoscopic full-thickness biopsy of the gastric wall with defect closure by using an endoscopic suturing device: survival porcine study.

Age sensitizes the kidney to heme protein-induced acute kidney injury.

Intractable nausea and vomiting from autoantibodies against a brain water channel.

Kit Signaling Is Required for Development of Coordinated Motility Patterns in Zebrafish Gastrointestinal Tract.

Complex Interactions Among Diet, Gastrointestinal Transit, and Gut Microbiota in Humanized Mice.

Cholecystectomy and Clinical Presentations of Gastroparesis.

Introducing our Associates.