David M. Bader

Hole is a novel gene product expressed in the developing heart and brain.

Development of the coronary vessel system.

Epicardial/Mesothelial cell line retains vasculogenic potential of embryonic epicardium.

Membrane topology of Bves/Pop1A, a cell adhesion molecule that displays dynamic changes in cellular distribution during development.

Bves expression during avian embryogenesis.

Bves is expressed in the epithelial components of the retina, lens, and cornea.

Cytoplasmic LEK1 is a regulator of microtubule function through its interaction with the LIS1 pathway.

Bves modulates epithelial integrity through an interaction at the tight junction.

The serosal mesothelium is a major source of smooth muscle cells of the gut vasculature.

Bves, a member of the Popeye domain-containing gene family.

Characterization of Bves expression during mouse development using newly generated immunoreagents.

CytLEK1 is a regulator of plasma membrane recycling through its interaction with SNAP-25.

Pancreatic islet production of vascular endothelial growth factor--a is essential for islet vascularization, revascularization, and function.

Signals from both sides: Control of cardiac development by the endocardium and epicardium.

Serosal mesothelium retains vasculogenic potential.

Identification of a novel intracellular interaction domain essential for Bves function.

Murine CENPF interacts with syntaxin 4 in the regulation of vesicular transport.

Expression of active Notch1 in avian coronary development.

Murine CENP-F regulates centrosomal microtubule nucleation and interacts with Hook2 at the centrosome.

Identification of a novel Bves function: regulation of vesicular transport.

Bves modulates tight junction associated signaling.

Application of small organic molecules reveals cooperative TGFβ and BMP regulation of mesothelial cell behaviors.

Omental grafting: a cell-based therapy for blood vessel repair.

Cardiac-specific deletion of the microtubule-binding protein CENP-F causes dilated cardiomyopathy.

Identification of a novel developmental mechanism in the generation of mesothelia.

Comprehensive timeline of mesodermal development in the quail small intestine.

Thymosin β4 mobilizes mesothelial cells for blood vessel repair.

Autotaxin signaling governs phenotypic heterogeneity in visceral and parietal mesothelia.

Bves and NDRG4 regulate directional epicardial cell migration through autocrine extracellular matrix deposition.

The chick embryo as an expanding experimental model for cancer and cardiovascular research.

Embryonic domains of the aorta derived from diverse origins exhibit distinct properties that converge into a common phenotype in the adult.

Resident progenitors, not exogenous migratory cells, generate the majority of visceral mesothelium in organogenesis.