A. Sue Menko

Role for alpha 6 integrin during lens development: Evidence for signaling through IGF-1R and ERK.

The role of Src family kinases in cortical cataract formation.

Regulation of cadherin junctions during mouse submandibular gland development.

Transition between proliferation and differentiation for lens epithelial cells is regulated by Src family kinases.

A signaling role for the uncleaved form of alpha 6 integrin in differentiating lens fiber cells.

Coordinate signaling by Src and p38 kinases in the induction of cortical cataracts.

Color image acquisition using a monochrome camera and standard fluorescence filter cubes.

The canonical intrinsic mitochondrial death pathway has a non-apoptotic role in signaling lens cell differentiation.

Actin filament organization regulates the induction of lens cell differentiation and survival.

Phosphatidylinositol 3-kinase is necessary for lens fiber cell differentiation and survival.

Activation of SRC kinases signals induction of posterior capsule opacification.

Mechanism of Src kinase induction of cortical cataract following exposure to stress: destabilization of cell-cell junctions.

Mechanism of small heat shock protein function in vivo: a knock-in mouse model demonstrates that the R49C mutation in alpha A-crystallin enhances protein insolubility and cell death.

Identification of a novel intermediate filament-linked N-cadherin/gamma-catenin complex involved in the establishment of the cytoarchitecture of differentiated lens fiber cells.

Integrins in lens development and disease.

Diverse roles of E-cadherin in the morphogenesis of the submandibular gland: insights into the formation of acinar and ductal structures.

Overexpression of DPAGT1 leads to aberrant N-glycosylation of E-cadherin and cellular discohesion in oral cancer.

Noggin producing, MyoD-positive cells are crucial for eye development.

Unique precursors for the mesenchymal cells involved in injury response and fibrosis.

αA-Crystallin associates with α6 integrin receptor complexes and regulates cellular signaling.

Modulation of N-cadherin junctions and their role as epicenters of differentiation-specific actin regulation in the developing lens.

Insulin-like growth factor receptor-1 and nuclear factor κB are crucial survival signals that regulate caspase-3-mediated lens epithelial cell differentiation initiation.

Distinct roles for N-Cadherin linked c-Src and fyn kinases in lens development.

Autophagy and mitophagy participate in ocular lens organelle degradation.

The Hippo signaling pathway is required for salivary gland development and its dysregulation is associated with Sjogren's syndrome.

α6 integrin transactivates insulin-like growth factor receptor-1 (IGF-1R) to regulate caspase-3-mediated lens epithelial cell differentiation initiation.

Cytokine deposition alters leukocyte morphology and initial recruitment of monocytes and γδT cells after corneal injury.

Endogenous hydrogen peroxide production in the epithelium of the developing embryonic lens.

Suppression of MAPK/JNK-MTORC1 signaling leads to premature loss of organelles and nuclei by autophagy during terminal differentiation of lens fiber cells.

Differentiation state-specific mitochondrial dynamic regulatory networks are revealed by global transcriptional analysis of the developing chicken lens.