Ron Balczon

Department of Biochemistry and Molecular Biology,
Center for Lung Biology

University of South Alabama

BIOGRAPHY

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PUBLICATIONS

Analysis of detached human kinetochores.

Chromosoma Aug, 2003 | Pubmed ID: 12883946

Stat3 activity is required for centrosome duplication in chinese hamster ovary cells.

The Journal of biological chemistry Oct, 2004 | Pubmed ID: 15294906

Soluble adenylyl cyclase-dependent microtubule disassembly reveals a novel mechanism of endothelial cell retraction.

American journal of physiology. Lung cellular and molecular physiology Jul, 2009 | Pubmed ID: 19395666

Cold exposure reveals two populations of microtubules in pulmonary endothelia.

American journal of physiology. Lung cellular and molecular physiology Jan, 2011 | Pubmed ID: 20971804

Filamin A is a phosphorylation target of membrane but not cytosolic adenylyl cyclase activity.

American journal of physiology. Lung cellular and molecular physiology Jul, 2011 | Pubmed ID: 21478251

Human pulmonary microvascular endothelial cells support productive replication of highly pathogenic avian influenza viruses: possible involvement in the pathogenesis of human H5N1 virus infection.

Journal of virology Jan, 2012 | Pubmed ID: 22072765

Pseudomonas aeruginosa exotoxin Y is a promiscuous cyclase that increases endothelial tau phosphorylation and permeability.

The Journal of biological chemistry Jul, 2012 | Pubmed ID: 22637478

Pseudomonas aeruginosa exotoxin Y-mediated tau hyperphosphorylation impairs microtubule assembly in pulmonary microvascular endothelial cells.

PloS one , 2013 | Pubmed ID: 24023939

The Pseudomonas aeruginosa exoenzyme Y impairs endothelial cell proliferation and vascular repair following lung injury.

American journal of physiology. Lung cellular and molecular physiology May, 2014 | Pubmed ID: 24705722

Pseudomonas aeruginosa exoenzymes U and Y induce a transmissible endothelial proteinopathy.

American journal of physiology. Lung cellular and molecular physiology Feb, 2016 | Pubmed ID: 26637633

The Pseudomonas aeruginosa Exoenzyme Y: A Promiscuous Nucleotidyl Cyclase Edema Factor and Virulence Determinant.

Handbook of experimental pharmacology , 2017 | Pubmed ID: 28181005

infection liberates transmissible, cytotoxic prion amyloids.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology 07, 2017 | Pubmed ID: 28314768

INSTITUTIONS

University of South Alabama

JOVE ARTICLES

Methods for Detecting Cytotoxic Amyloids Following Infection of Pulmonary Endothelial Cells by Pseudomonas aeruginosa

Ron Balczon^1,4,

Michael Francis^2,4,

Silas Leavesley^3,4,

Troy Stevens^2,4

¹Department of Biochemistry and Molecular Biology, University of South Alabama,

²Department of Physiology and Cell Biology, University of South Alabama,

³Department of Chemical and Biomolecular Engineering, University of South Alabama,

⁴Center for Lung Biology, University of South Alabama

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Ron Balczon

.css-o250i3{font-size:18px;font-family:Open Sans,sans-serif;line-height:21.6px;}Department of Biochemistry and Molecular Biology,Center for Lung Biology

University of South Alabama

Methods for Detecting Cytotoxic Amyloids Following Infection of Pulmonary Endothelial Cells by Pseudomonas aeruginosa

Department of Biochemistry and Molecular Biology,
Center for Lung Biology