Ron Balczon

Analysis of detached human kinetochores.

Stat3 activity is required for centrosome duplication in chinese hamster ovary cells.

Soluble adenylyl cyclase-dependent microtubule disassembly reveals a novel mechanism of endothelial cell retraction.

Cold exposure reveals two populations of microtubules in pulmonary endothelia.

Filamin A is a phosphorylation target of membrane but not cytosolic adenylyl cyclase activity.

Human pulmonary microvascular endothelial cells support productive replication of highly pathogenic avian influenza viruses: possible involvement in the pathogenesis of human H5N1 virus infection.

Pseudomonas aeruginosa exotoxin Y is a promiscuous cyclase that increases endothelial tau phosphorylation and permeability.

Pseudomonas aeruginosa exotoxin Y-mediated tau hyperphosphorylation impairs microtubule assembly in pulmonary microvascular endothelial cells.

The Pseudomonas aeruginosa exoenzyme Y impairs endothelial cell proliferation and vascular repair following lung injury.

Pseudomonas aeruginosa exoenzymes U and Y induce a transmissible endothelial proteinopathy.

The Pseudomonas aeruginosa Exoenzyme Y: A Promiscuous Nucleotidyl Cyclase Edema Factor and Virulence Determinant.

infection liberates transmissible, cytotoxic prion amyloids.