Hiroshi Kitani

Recombinant tumor necrosis factor alpha does not inhibit the growth of African trypanosomes in axenic cultures.

Evaluation of the delta subunit of bovine adaptor protein complex 3 as a receptor for bovine leukaemia virus.

The secretion of acute phase proteins and inflammatory cytokines during Trypanosoma congolense infection is not affected by the absence of the TNF-alpha gene.

Susceptibility of TNF-alpha-deficient mice to Trypanosoma congolense is not due to a defective antibody response.

Inhibitory effects of U73122 and U73343 on Ca2+ influx and pore formation induced by the activation of P2X7 nucleotide receptors in mouse microglial cell line.

Intrabodies against the EVH1 domain of Wiskott-Aldrich syndrome protein inhibit T cell receptor signaling in transgenic mice T cells.

Protective effects of antimicrobial peptides derived from the beetle Allomyrina dichotoma defensin on endotoxic shock in mice.

Mouse embryonic stem cells are not susceptible to cytomegalovirus but acquire susceptibility during differentiation.

Suppressive effect of simvastatin on interferon-beta-induced expression of CC chemokine ligand 5 in microglia.

Establishment and characterization of SV40 large T antigen-immortalized cell lines derived from fetal bovine brain tissues after prolonged cryopreservation.

Microglial cell line established from prion protein-overexpressing mice is susceptible to various murine prion strains.

Roles of NF-kappaB and MAPK signaling pathways in morphological and cytoskeletal responses of microglia to double-stranded RNA.

Lysophosphatidylcholine potentiates Ca2+ influx, pore formation and p44/42 MAP kinase phosphorylation mediated by P2X7 receptor activation in mouse microglial cells.

Prion infection correlates with hypersensitivity of P2X7 nucleotide receptor in a mouse microglial cell line.

Impaired LPS-induced signaling in microglia overexpressing the Wiskott-Aldrich syndrome protein N-terminal domain.

Activin as an anti-inflammatory cytokine produced by microglia.

Lysophospholipids and ATP mutually suppress maturation and release of IL-1 beta in mouse microglial cells using a Rho-dependent pathway.

[Establishment and characterization of prion-infected microglial cells].

Lactoferrin induces cell surface retention of prion protein and inhibits prion accumulation.

Establishment of an SV40 large T antigen-immortalized bovine brain cell line and its neuronal differentiation by dibutyryl-cyclic AMP.

Cytoplasmic expression and specific binding of the VH/VL single domain intrabodies in transfected NIH3T3 cells.

Microglial activation is inhibited by corticosterone in dopaminergic neurodegeneration.

The activation of P2X7 receptor impairs lysosomal functions and stimulates the release of autophagolysosomes in microglial cells.

Synthetic nonamer peptides derived from insect defensin mediate the killing of African trypanosomes in axenic culture.

The role of the P2X7 receptor signaling pathway for the release of autolysosomes in microglial cells.

Characteristics of novel insect defensin-based membrane-disrupting trypanocidal peptides.

Modulation of the ATP-lnduced release and processing of IL-1beta in microglial cells.

Possible roles of microglial cells for neurotoxicity in clinical neurodegenerative diseases and experimental animal models.

gamma-Irradiation induces P2X(7) receptor-dependent ATP release from B16 melanoma cells.

P2X7 receptor signaling pathway as a therapeutic target for neurodegenerative diseases.

Celastrol suppresses morphological and transcriptional responses in microglial cells upon stimulation with double-stranded RNA.

A novel isolation method for macrophage-like cells from mixed primary cultures of adult rat liver cells.

Enhancement of phagocytotic activity by prion protein in PrP-deficient macrophage cells.

Cold stress induced morphological microglial activation and increased IL-1β expression in astroglial cells in rat brain.

Angiopoietin-1/Tie2 signal augments basal Notch signal controlling vascular quiescence by inducing delta-like 4 expression through AKT-mediated activation of beta-catenin.

Isolation and characterization of macrophages from a mixed primary culture of bovine liver cells.

The activation of P2X7 receptor induces cathepsin D-dependent production of a 20-kDa form of IL-1β under acidic extracellular pH in LPS-primed microglial cells.

Identification of Fyn as the binding partner for the WASP N-terminal domain in T cells.

PLD$ is involved in phagocytosis of microglia: expression and localization changes of PLD4 are correlated with activation state of microglia.

Carnosic acid, a pro-electrophilic compound, inhibits LPS-induced activation of microglia.

Critical Roles of the WASP N-Terminal Domain and Btk in LPS-Induced Inflammatory Response in Macrophages.