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University Paris Descartes Hospital Necker-Enfants Malades
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FUS and TARDBP but not SOD1 interact in genetic models of amyotrophic lateral sclerosis.
PLoS genetics Aug, 2011 | Pubmed ID: 21829392
Defining the association of TMEM106B variants among frontotemporal lobar degeneration patients with GRN mutations and C9orf72 repeat expansions.
Neurobiology of aging Nov, 2014 | Pubmed ID: 25085782
Contribution of ATXN2 intermediary polyQ expansions in a spectrum of neurodegenerative disorders.
Neurology Sep, 2014 | Pubmed ID: 25098532
Sqstm1 knock-down causes a locomotor phenotype ameliorated by rapamycin in a zebrafish model of ALS/FTLD.
Human molecular genetics Mar, 2015 | Pubmed ID: 25410659
Defining the genetic connection linking amyotrophic lateral sclerosis (ALS) with frontotemporal dementia (FTD).
Trends in genetics : TIG May, 2015 | Pubmed ID: 25869998
Depdc5 knockdown causes mTOR-dependent motor hyperactivity in zebrafish.
Annals of clinical and translational neurology May, 2018 | Pubmed ID: 29761115
Transcriptomic Analysis of Zebrafish TDP-43 Transgenic Lines.
Frontiers in molecular neuroscience , 2018 | Pubmed ID: 30618614
Functional Characterization of Neurofilament Light Splicing and Misbalance in Zebrafish.
Cells 05, 2020 | Pubmed ID: 32429483
TDP-43 Regulation of AChE Expression Can Mediate ALS-Like Phenotype in Zebrafish.
Cells 01, 2021 | Pubmed ID: 33499374
Autophagy and ALS: mechanistic insights and therapeutic implications.
Autophagy May, 2021 | Pubmed ID: 34057020
institut imagine
Sorbonne Universités Paris
Hortense de Calbiac*,1,2,
Adriana Dabacan*,3,
Raul Muresan3,
Edor Kabashi1,2,
Sorana Ciura1,2
1University Paris Descartes Hospital Necker-Enfants Malades, Institut Imagine,
2Institut du Cerveau et de la Moelle épinière - ICM, Sorbonne Universités Paris,
3, Transylvanian Institute of Neuroscience (TINS)
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