Benno Roozendaal

Glucocorticoids interact with the basolateral amygdala beta-adrenoceptor--cAMP/cAMP/PKA system in influencing memory consolidation.

Basolateral amygdala lesions block the memory-enhancing effect of 8-Br-cAMP infused into the entorhinal cortex of rats after training.

Role of adrenal stress hormones in forming lasting memories in the brain.

Involvement of stress-released corticotropin-releasing hormone in the basolateral amygdala in regulating memory consolidation.

The hippocampus mediates glucocorticoid-induced impairment of spatial memory retrieval: dependence on the basolateral amygdala.

Stress and memory: opposing effects of glucocorticoids on memory consolidation and memory retrieval.

Glucocorticoid-induced impairment of declarative memory retrieval is associated with reduced blood flow in the medial temporal lobe.

Role of the basolateral amygdala in memory consolidation.

Exposure to high stress in the intensive care unit may have negative effects on health-related quality-of-life outcomes after cardiac surgery.

Systems mediating acute glucocorticoid effects on memory consolidation and retrieval.

Memory enhancement of classical fear conditioning by post-training injections of corticosterone in rats.

Glucocorticoid effects on object recognition memory require training-associated emotional arousal.

The basolateral amygdala interacts with the medial prefrontal cortex in regulating glucocorticoid effects on working memory impairment.

A systemically administered beta-adrenoceptor antagonist blocks corticosterone-induced impairment of contextual memory retrieval in rats.

Stress doses of hydrocortisone, traumatic memories, and symptoms of posttraumatic stress disorder in patients after cardiac surgery: a randomized study.

Low-dose cortisol for symptoms of posttraumatic stress disorder.

Glucocorticoid effects on memory retrieval require concurrent noradrenergic activity in the hippocampus and basolateral amygdala.

Glucocorticoid therapy and memory function: lessons learned from basic research.

Can posttraumatic stress disorder be prevented with glucocorticoids?

Basolateral amygdala interacts with other brain regions in regulating glucocorticoid effects on different memory functions.

Dexamethasone reverses the memory impairment induced by antagonism of hippocampal gastrin-releasing peptide receptors.

Insular cortex is involved in consolidation of object recognition memory.

Relaxin receptor activation in the basolateral amygdala impairs memory consolidation.

Stress doses of hydrocortisone reduce chronic stress symptoms and improve health-related quality of life in high-risk patients after cardiac surgery: a randomized study.

Glucocorticoids reduce phobic fear in humans.

Glucocorticoid enhancement of memory requires arousal-induced noradrenergic activation in the basolateral amygdala.

Basolateral amygdala noradrenergic activity mediates corticosterone-induced enhancement of auditory fear conditioning.

Posttraining handling facilitates memory for auditory-cue fear conditioning in rats.

Efficacy of hydrocortisone in preventing posttraumatic stress disorder following critical illness and major surgery.

Glucocorticoids increase amyloid-beta and tau pathology in a mouse model of Alzheimer's disease.

Orphanin FQ/nociceptin interacts with the basolateral amygdala noradrenergic system in memory consolidation.

Preventive effect of beta-adrenoceptor blockade on glucocorticoid-induced memory retrieval deficits.

Glucocorticoid administration into the dorsal striatum [corrected] facilitates memory consolidation of inhibitory avoidance training but not of the context or footshock components.

Adrenal stress hormones, amygdala activation, and memory for emotionally arousing experiences.

Glucocorticoid therapy-induced memory deficits: acute versus chronic effects.

Corticotropin-releasing factor in the basolateral amygdala enhances memory consolidation via an interaction with the beta-adrenoceptor-cAMP pathway: dependence on glucocorticoid receptor activation.

Glucocorticoids enhance taste aversion memory via actions in the insular cortex and basolateral amygdala.

Noradrenergic activation of the basolateral amygdala modulates consolidation of object recognition memory.

Drug enhancement of memory consolidation: historical perspective and neurobiological implications.

Endocannabinoids in the rat basolateral amygdala enhance memory consolidation and enable glucocorticoid modulation of memory.

Glucocorticoids and the regulation of memory in health and disease.

Traumatic memories, post-traumatic stress disorder and serum cortisol levels in long-term survivors of the acute respiratory distress syndrome.

Fat-induced satiety factor oleoylethanolamide enhances memory consolidation.

Stress, memory and the amygdala.

Interacting noradrenergic and corticosteroid systems shift human brain activation patterns during encoding.

Corticosterone infused into the dorsal striatum selectively enhances memory consolidation of cued water-maze training.

Glucocorticoid effects on memory consolidation depend on functional interactions between the medial prefrontal cortex and basolateral amygdala.

Memory-enhancing corticosterone treatment increases amygdala norepinephrine and Arc protein expression in hippocampal synaptic fractions.

Membrane-associated glucocorticoid activity is necessary for modulation of long-term memory via chromatin modification.

Glucocorticoids in the prefrontal cortex enhance memory consolidation and impair working memory by a common neural mechanism.

Relationship of a common polymorphism of the glucocorticoid receptor gene to traumatic memories and posttraumatic stress disorder in patients after intensive care therapy.

Propofol enhances memory formation via an interaction with the endocannabinoid system.

Stress and emotional memory: a matter of timing.

The cortisol awakening response in amyotrophic lateral sclerosis is blunted and correlates with clinical status and depressive mood.

Experience modulates vicarious freezing in rats: a model for empathy.

Stress effects on memory: An update and integration.

Memory modulation.

Glucocorticoids interact with the hippocampal endocannabinoid system in impairing retrieval of contextual fear memory.