Jianxin Wu

Effects of mechanical stress and carvedilol in lamin A/C-deficient dilated cardiomyopathy.

Regulation of murine cardiac contractility by activation of α(1A)-adrenergic receptor-operated Ca(2+) entry.

A proliferative burst during preadolescence establishes the final cardiomyocyte number.

Pressure Overload by Transverse Aortic Constriction Induces Maladaptive Hypertrophy in a Titin-Truncated Mouse Model.

Orphan receptor GPR37L1 contributes to the sexual dimorphism of central cardiovascular control.

Cardiac hypertrophy limits infarct expansion after myocardial infarction in mice.

Pressure overload by suprarenal aortic constriction in mice leads to left ventricular hypertrophy without c-Kit expression in cardiomyocytes.

Adaptation to exercise-induced stress is not dependent on cardiomyocyte α-adrenergic receptors.

Cardiac Gq Receptors and Calcineurin Activation Are Not Required for the Hypertrophic Response to Mechanical Left Ventricular Pressure Overload.

The Ca-activated cation channel TRPM4 is a positive regulator of pressure overload-induced cardiac hypertrophy.

Involvement of GPR37L1 in murine blood pressure regulation and human cardiac disease pathophysiology.