Carlo Perego

Functional role of inflammatory cytokines and antiinflammatory molecules in seizures and epileptogenesis.

Framework topology of ERS-10 zeolite.

Prophylactic but not delayed administration of simvastatin protects against long-lasting cognitive and morphological consequences of neonatal hypoxic-ischemic brain injury, reduces interleukin-1beta and tumor necrosis factor-alpha mRNA induction, and does not affect endothelial nitric oxide synthase expression.

Glia activation and cytokine increase in rat hippocampus by kainic acid-induced status epilepticus during postnatal development.

Peripheral treatment with enoxaparin, a low molecular weight heparin, reduces plaques and beta-amyloid accumulation in a mouse model of Alzheimer's disease.

Pentoxifylline prevents spontaneous brain ischemia in stroke-prone rats.

Functional role of proinflammatory and anti-inflammatory cytokines in seizures.

Tumor necrosis factor-alpha inhibits seizures in mice via p75 receptors.

Inflammatory response and glia activation in developing rat hippocampus after status epilepticus.

Simvastatin reduces caspase-3 activation and inflammatory markers induced by hypoxia-ischemia in the newborn rat.

C1-inhibitor attenuates neurobehavioral deficits and reduces contusion volume after controlled cortical impact brain injury in mice.

c-Jun N-terminal kinase pathway activation in human and experimental cerebral contusion.

New method for H(2)S removal in acid solutions.

Glycogen synthase kinase-3 inhibition reduces ischemic cerebral damage, restores impaired mitochondrial biogenesis and prevents ROS production.

Long-lasting protection in brain trauma by endotoxin preconditioning.

CX3CL1 is neuroprotective in permanent focal cerebral ischemia in rodents.

Temporal pattern of expression and colocalization of microglia/macrophage phenotype markers following brain ischemic injury in mice.

Porous materials in catalysis: challenges for mesoporous materials.

CX3CR1 deficiency induces an early protective inflammatory environment in ischemic mice.

Tumor necrosis factor in traumatic brain injury: effects of genetic deletion of p55 or p75 receptor.