Beat A. Imhof

Hyperglycemia-induced vasculopathy in the murine vitelline vasculature: correlation with PECAM-1/CD31 tyrosine phosphorylation state.

Forging the endothelium during inflammation: pushing at a half-open door?

Expression and function of junctional adhesion molecule-C in human and experimental arthritis.

Expression and function of junctional adhesion molecule-C in myelinated peripheral nerves.

Adaptive immune response in JAM-C-deficient mice: normal initiation but reduced IgG memory.

Dimerization of Kit-ligand and efficient cell-surface presentation requires a conserved Ser-Gly-Gly-Tyr motif in its transmembrane domain.

Junctional adhesion molecule-C mediates leukocyte infiltration in response to ischemia reperfusion injury.

Tumour angiogenesis is reduced in the Tc1 mouse model of Down's syndrome.

Targeting vascular NADPH oxidase 1 blocks tumor angiogenesis through a PPARα mediated mechanism.

Cadm1-expressing synapses on Purkinje cell dendrites are involved in mouse ultrasonic vocalization activity.

Targeting olfactomedin-like 3 inhibits tumor growth by impairing angiogenesis and pericyte coverage.

Junctional adhesion molecule (JAM)-C deficient C57BL/6 mice develop a severe hydrocephalus.

Three-dimensional visualization of the mouse thymus organization in health and immunodeficiency.

Antigen recognition by autoreactive CD4⁺ thymocytes drives homeostasis of the thymic medulla.

Thymic epithelial cell expansion through matricellular protein CYR61 boosts progenitor homing and T-cell output.

Abnormal junctions and permeability of myelin in PMP22-deficient nerves.

VE-cadherin phosphorylation decides: vascular permeability or diapedesis.

Matricellular protein CCN1/CYR61: a new player in inflammation and leukocyte trafficking.

Introduction for the special issue on new paradigms in leukocyte trafficking, lessons for therapeutics.

Blocking junctional adhesion molecule C enhances dendritic cell migration and boosts the immune responses against Leishmania major.

5'-Ectonucleotidase/CD73 expression on lymph-circulating lymphocytes and lymphatic endothelial cells offers new paths to explore barrier function.

Junctional adhesion molecule B interferes with angiogenic VEGF/VEGFR2 signaling.

Reverse Migration of Neutrophils: Where, When, How, and Why?

Murine junctional adhesion molecules JAM-B and JAM-C mediate endothelial and stellate cell interactions during hepatic fibrosis.

Anti-JAM-C therapy eliminates tumor engraftment in a xenograft model of mantle cell lymphoma.

Divergent JAM-C Expression Accelerates Monocyte-Derived Cell Exit from Atherosclerotic Plaques.

Élie Metchnikoff (1845-1916): celebrating 100 years of cellular immunology and beyond.

CCN1/CYR61-mediated meticulous patrolling by Ly6Clow monocytes fuels vascular inflammation.

Biodegradable and plasma-treated electrospun scaffolds coated with recombinant Olfactomedin-like 3 for accelerating wound healing and tissue regeneration.

Aortic aneurysm, CCN3 may solve the problem.

Monocyte Subsets Coregulate Inflammatory Responses by Integrated Signaling through TNF and IL-6 at the Endothelial Cell Interface.

Toll-like receptors elicit different recruitment kinetics of monocytes and neutrophils in mouse acute inflammation.

Design, synthesis and biological evaluation of new pyrazolyl-ureas and imidazopyrazolecarboxamides able to interfere with MAPK and PI3K upstream signaling involved in the angiogenesis.

The pyrazolyl-urea GeGe3 inhibits tumor angiogenesis and reveals dystrophia myotonica protein kinase (DMPK)1 as a novel angiogenesis target.

Angiogenic factor-driven inflammation promotes extravasation of human proangiogenic monocytes to tumours.

Junctional adhesion molecule C (JAM-C) dimerization aids cancer cell migration and metastasis.

Junctional adhesion molecules JAM-B and JAM-C promote autoimmune-mediated liver fibrosis in mice.