Karl W. Kafitz

Postsynaptic Induction of BDNF-Mediated Long-Term Potentiation.

Neurotrophin-evoked depolarization requires the sodium channel Na(V)1.9.

Glomerular territories in the olfactory bulb from the larval stage of the sea lamprey Petromyzon marinus.

Truncated TrkB-T1 mediates neurotrophin-evoked calcium signalling in glia cells.

From modulator to mediator: rapid effects of BDNF on ion channels.

Developmental profile and mechanisms of GABA-induced calcium signaling in hippocampal astrocytes.

Sodium signals in cerebellar Purkinje neurons and Bergmann glial cells evoked by glutamatergic synaptic transmission.

Ammonium-evoked alterations in intracellular sodium and pH reduce glial glutamate transport activity.

Astrocyte calcium signals at Schaffer collateral to CA1 pyramidal cell synapses correlate with the number of activated synapses but not with synaptic strength.

Kir4.1 channels mediate a depolarization of hippocampal astrocytes under hyperammonemic conditions in situ.

Activation of CRH receptor type 1 expressed on glutamatergic neurons increases excitability of CA1 pyramidal neurons by the modulation of voltage-gated ion channels.

Laminar and subcellular heterogeneity of GLAST and GLT-1 immunoreactivity in the developing postnatal mouse hippocampus.

Lesion-induced alterations in astrocyte glutamate transporter expression and function in the hippocampus.

Extrusion versus diffusion: mechanisms for recovery from sodium loads in mouse CA1 pyramidal neurons.

Changes in the proliferative capacity of NG2 cell subpopulations during postnatal development of the mouse hippocampus.

Rapid sodium signaling couples glutamate uptake to breakdown of ATP in perivascular astrocyte endfeet.

Astrocyte Sodium Signalling and Panglial Spread of Sodium Signals in Brain White Matter.

FRET-based imaging of intracellular ATP in organotypic brain slices.

Imaging of Local and Global Sodium Signals in Astrocytes.

Dysregulation of Astrocyte Ion Homeostasis and Its Relevance for Stroke-Induced Brain Damage.