Troy Stevens

Essential control of an endothelial cell ISOC by the spectrin membrane skeleton.

Dominant regulation of interendothelial cell gap formation by calcium-inhibited type 6 adenylyl cyclase.

Coordinate regulation of membrane cAMP by Ca2+-inhibited adenylyl cyclase and phosphodiesterase activities.

On the endothelial cell I(SOC).

Cav3.1 (alpha1G) T-type Ca2+ channels mediate vaso-occlusion of sickled erythrocytes in lung microcirculation.

Structural and functional characteristics of lung macro- and microvascular endothelial cell phenotypes.

Potential role for activated leukocyte cell adhesion molecule and neural cadherin in metastasis to the lung microcirculation.

Paradoxical cAMP-induced lung endothelial hyperpermeability revealed by Pseudomonas aeruginosa ExoY.

Stat3 activity is required for centrosome duplication in chinese hamster ovary cells.

Activated leukocyte cell adhesion molecule in breast cancer: prognostic indicator.

Essential role of a Ca2+-selective, store-operated current (ISOC) in endothelial cell permeability: determinants of the vascular leak site.

Activation of the endothelial store-operated ISOC Ca2+ channel requires interaction of protein 4.1 with TRPC4.

Molecular and cellular determinants of lung endothelial cell heterogeneity.

Soluble adenylyl cyclase reveals the significance of cAMP compartmentation on pulmonary microvascular endothelial cell barrier.

Activated leukocyte cell adhesion molecule is a component of the endothelial junction involved in transendothelial monocyte migration.

Regulation of endothelial cell barrier function by store-operated calcium entry.

Microtubule motors regulate ISOC activation necessary to increase endothelial cell permeability.

Phenotypic heterogeneity in lung capillary and extra-alveolar endothelial cells. Increased extra-alveolar endothelial permeability is sufficient to decrease compliance.

Regulatory role for nucleosome assembly protein-1 in the proliferative and vasculogenic phenotype of pulmonary endothelium.

Hydroxyurea attenuates activated neutrophil-mediated sickle erythrocyte membrane phosphatidylserine exposure and adhesion to pulmonary vascular endothelium.

Lung microvascular endothelium is enriched with progenitor cells that exhibit vasculogenic capacity.

Heterogeneity of barrier function in the lung reflects diversity in endothelial cell junctions.

Spectrin-anchored phosphodiesterase 4D4 restricts cAMP from disrupting microtubules and inducing endothelial cell gap formation.

The cancer paradigm of severe pulmonary arterial hypertension.

Lung vascular cell heterogeneity: endothelium, smooth muscle, and fibroblasts.

TRPing on the lung endothelium: calcium channels that regulate barrier function.

The actin cytoskeleton in endothelial cell phenotypes.

Type 5 phosphodiesterase expression is a critical determinant of the endothelial cell angiogenic phenotype.

Soluble adenylyl cyclase-dependent microtubule disassembly reveals a novel mechanism of endothelial cell retraction.

Development and pathology of pulmonary hypertension.

Store-operated calcium entry channels in pulmonary endothelium: the emerging story of TRPCS and Orai1.

Perivascular fluid cuffs decrease lung compliance by increasing tissue resistance.

New developments in lung endothelial heterogeneity: Von Willebrand factor, P-selectin, and the Weibel-Palade body.

Protein kinase A phosphorylation of tau-serine 214 reorganizes microtubules and disrupts the endothelial cell barrier.

Critical role for lactate dehydrogenase A in aerobic glycolysis that sustains pulmonary microvascular endothelial cell proliferation.

Cold exposure reveals two populations of microtubules in pulmonary endothelia.

Filamin A is a phosphorylation target of membrane but not cytosolic adenylyl cyclase activity.

Adenylyl and guanylyl cyclase assays.

Studies on the cell biology of interendothelial cell gaps.

Functional and molecular heterogeneity of pulmonary endothelial cells.

Human pulmonary microvascular endothelial cells support productive replication of highly pathogenic avian influenza viruses: possible involvement in the pathogenesis of human H5N1 virus infection.

Orai1 determines calcium selectivity of an endogenous TRPC heterotetramer channel.

Pseudomonas aeruginosa exotoxin Y is a promiscuous cyclase that increases endothelial tau phosphorylation and permeability.

Pseudomonas aeruginosa exotoxin Y-mediated tau hyperphosphorylation impairs microtubule assembly in pulmonary microvascular endothelial cells.

Lactate dehydrogenase a expression is necessary to sustain rapid angiogenesis of pulmonary microvascular endothelium.

TRPC4 inactivation confers a survival benefit in severe pulmonary arterial hypertension.

Studies on the resolution of subcellular free calcium concentrations: a technological advance. Focus on "detection of differentially regulated subsarcolemmal calcium signals activated by vasoactive agonists in rat pulmonary artery smooth muscle cells".

The Pseudomonas aeruginosa exoenzyme Y impairs endothelial cell proliferation and vascular repair following lung injury.

A unique pulmonary microvascular endothelial cell niche revealed by Weibel-Palade bodies and Griffonia simplicifolia.

Sodium entry through endothelial store-operated calcium entry channels: regulation by Orai1.

Heterogeneity of pulmonary endothelial cyclic nucleotide response to Pseudomonas aeruginosa ExoY infection.

Transmembrane proteoglycans control stretch-activated channels to set cytosolic calcium levels.

Pseudomonas aeruginosa exoenzymes U and Y induce a transmissible endothelial proteinopathy.

Transient Receptor Potential Channel 4 Encodes a Vascular Permeability Defect and High-Frequency Ca(2+) Transients in Severe Pulmonary Arterial Hypertension.

Endothelial hyperpermeability in severe pulmonary arterial hypertension: role of store-operated calcium entry.

"Small Blood Vessels: Big Health Problems?": Scientific Recommendations of the National Institutes of Health Workshop.

The Pseudomonas aeruginosa Exoenzyme Y: A Promiscuous Nucleotidyl Cyclase Edema Factor and Virulence Determinant.

infection liberates transmissible, cytotoxic prion amyloids.

Single cell cloning generates lung endothelial colonies with conserved growth, angiogenic, and bioenergetic characteristics.

Pulmonary vascular dysfunction secondary to pulmonary arterial hypertension: insights gained through retrograde perfusion.

Carbonic anhydrase IX is a critical determinant of pulmonary microvascular endothelial cell pH regulation and angiogenesis during acidosis.

The role of endothelial leak in pulmonary hypertension (2017 Grover Conference Series).

Nosocomial Pneumonia Elicits an Endothelial Proteinopathy: Evidence for a Source of Neurotoxic Amyloids in Critically Ill Patients.

Extrinsic acidosis suppresses glycolysis and migration while increasing network formation in pulmonary microvascular endothelial cells.

Infection-induced endothelial amyloids impair memory.

Mechanical signaling in a pulmonary microvascular endothelial cell monolayer.

Unleashing shear: Role of intercellular traction and cellular moments in collective cell migration.

A cancer amidst us: the plexiform lesion in pulmonary arterial hypertension.

α-Tocopherol Attenuates the Severity of -induced Pneumonia.

Virulent Pseudomonas aeruginosa infection converts antimicrobial amyloids into cytotoxic prions.

Exoenzyme Y Contributes to End-Organ Dysfunction Caused by Pneumonia in Critically Ill Patients: An Exploratory Study.

Pneumonia-induced endothelial amyloids reduce dendritic spine density in brain neurons.

Biventricular diastolic dysfunction, thrombocytopenia, and red blood cell macrocytosis in experimental pulmonary arterial hypertension.

Exoenzyme Y induces extracellular active caspase-7 accumulation independent from apoptosis: modulation of transmissible cytotoxicity.

Development of an endothelial cell-restricted transgenic reporter rat: a resource for physiological studies of vascular biology.

KD025 Shifts Pulmonary Endothelial Cell Bioenergetics and Decreases Baseline Lung Permeability.

Cystatin C regulates the cytotoxicity of infection-induced endothelial-derived β-amyloid.

Impact of Na+ permeation on collective migration of pulmonary arterial endothelial cells.

Salvaging the endothelium in acute respiratory distress syndrome: a druggable intersection between TLR4 and NAD signalling.

Endothelial metabolism in pulmonary vascular homeostasis and acute respiratory distress syndrome.

Carbonic Anhydrase IX and Hypoxia Promote Rat Pulmonary Endothelial Cell Survival during Infection.

Pneumonia initiates a tauopathy.

Cytotoxic tau released from lung microvascular endothelial cells upon infection with Pseudomonas aeruginosa promotes neuronal tauopathy.

ExoU Induces Lung Endothelial Cell Damage and Activates Pro-Inflammatory Caspase-1 during Infection.

Carbonic anhydrase IX proteoglycan-like and intracellular domains mediate pulmonary microvascular endothelial cell repair and angiogenesis.

New Insights into Clinical and Mechanistic Heterogeneity of the Acute Respiratory Distress Syndrome: Summary of the Aspen Lung Conference 2021.

Substrate stiffness modulates migration and local intercellular membrane motion in pulmonary endothelial cell monolayers.

PFKFB3 Inhibits Fructose Metabolism in Pulmonary Microvascular Endothelial Cells.