Weihong Song

Dopamine-dependent neurotoxicity of alpha-synuclein: a mechanism for selective neurodegeneration in Parkinson disease.

Transcriptional regulation of BACE1, the beta-amyloid precursor protein beta-secretase, by Sp1.

Degradation of BACE by the ubiquitin-proteasome pathway.

Mechanism of promoter activity of the beta-amyloid precursor protein gene in different cell lines: identification of a specific 30 bp fragment in the proximal promoter region.

BACE1 gene expression and protein degradation.

Distinct transcriptional regulation and function of the human BACE2 and BACE1 genes.

Control of APP processing and Abeta generation level by BACE1 enzymatic activity and transcription.

Leaky scanning and reinitiation regulate BACE1 gene expression.

Increased BACE1 maturation contributes to the pathogenesis of Alzheimer's disease in Down syndrome.

BACE2, as a novel APP theta-secretase, is not responsible for the pathogenesis of Alzheimer's disease in Down syndrome.

Ubiquitin-proteasome pathway mediates degradation of APH-1.

Hypoxia facilitates Alzheimer's disease pathogenesis by up-regulating BACE1 gene expression.

Recent advances in basic neurosciences and brain disease: from synapses to behavior.

The cholesterol transporter ABCG1 modulates the subcellular distribution and proteolytic processing of beta-amyloid precursor protein.

Degradation of nicastrin involves both proteasome and lysosome.

SP1 regulates a human SNAP-25 gene expression.

Evidence that gamma-secretase mediates oxidative stress-induced beta-secretase expression in Alzheimer's disease.

Valproic acid inhibits Abeta production, neuritic plaque formation, and behavioral deficits in Alzheimer's disease mouse models.

TMP21 degradation is mediated by the ubiquitin-proteasome pathway.

Preparation of PbS Nanoparticles by Phase-Transfer Method and Application to Pb-Selective Electrode Based on PVC Membrane.

Upregulation of macrophage migration inhibitory factor gene expression in stroke.

Degradation of regulator of calcineurin 1 (RCAN1) is mediated by both chaperone-mediated autophagy and ubiquitin proteasome pathways.

BACE1 gene promoter single-nucleotide polymorphisms in Alzheimer's disease.

NF-κB signaling inhibits ubiquitin carboxyl-terminal hydrolase L1 gene expression.

Regulator of calcineurin 1 (RCAN1) facilitates neuronal apoptosis through caspase-3 activation.

Increased NF-κB signalling up-regulates BACE1 expression and its therapeutic potential in Alzheimer's disease.

Transcriptional Regulation of TMP21 by NFAT.

Loss of activated CaMKII at the synapse underlies Alzheimer's disease memory loss.

Regulation of β-site APP-cleaving enzyme 1 gene expression and its role in Alzheimer's disease.

Control of BACE1 degradation and APP processing by ubiquitin carboxyl-terminal hydrolase L1.

低氧调节的 ATP13A2 (PARK9) 基因转录。