Felipe Court

Calcium release from intra-axonal endoplasmic reticulum leads to axon degeneration through mitochondrial dysfunction.

Altered potassium channel distribution and composition in myelinated axons suppresses hyperexcitability following injury.

Injury to the nervous system: A look into the ER.

Bursting the unfolded protein response accelerates axonal regeneration.

Origin of axonal proteins: Is the axon-schwann cell unit a functional syncytium?

Schwann Cell and Axon: An Interlaced Unit-From Action Potential to Phenotype Expression.

Herpes Simplex Virus Type 1 Neuronal Infection Perturbs Golgi Apparatus Integrity through Activation of Src Tyrosine Kinase and Dyn-2 GTPase.

Axonal Degeneration during Aging and Its Functional Role in Neurodegenerative Disorders.

Disulfide cross-linked multimers of TDP-43 and spinal motoneuron loss in a TDP-43 ALS/FTD mouse model.

Molecular analysis of axonal-intrinsic and glial-associated co-regulation of axon degeneration.

A dual role for Integrin α6β4 in modulating hereditary neuropathy with liability to pressure palsies.

In Vitro Analysis of the Role of Schwann Cells on Axonal Degeneration and Regeneration Using Sensory Neurons from Dorsal Root Ganglia.

Purification of Exosomes from Primary Schwann Cells, RNA Extraction, and Next-Generation Sequencing of Exosomal RNAs.

Teased Fiber Preparation of Myelinated Nerve Fibers from Peripheral Nerves for Vital Dye Staining and Immunofluorescence Analysis.

The inhibition of CTGF/CCN2 activity improves muscle and locomotor function in a murine ALS model.

IRE1α governs cytoskeleton remodelling and cell migration through a direct interaction with filamin A.

Mitochondria and Calcium Regulation as Basis of Neurodegeneration Associated With Aging.

Publisher Correction: IRE1α governs cytoskeleton remodelling and cell migration through a direct interaction with filamin A.

Axonal degeneration induced by glutamate excitotoxicity is mediated by necroptosis.

Axonal Degeneration Is Mediated by Necroptosis Activation.

Compartmentalized necroptosis activation in excitotoxicity-induced axonal degeneration: a novel mechanism implicated in neurodegenerative disease pathology.

Non-canonical function of IRE1α determines mitochondria-associated endoplasmic reticulum composition to control calcium transfer and bioenergetics.

Altered mitochondrial bioenergetics are responsible for the delay in Wallerian degeneration observed in neonatal mice.

Publisher Correction: Non-canonical function of IRE1α determines mitochondria-associated endoplasmic reticulum composition to control calcium transfer and bioenergetics.

The p75 neurotrophin receptor is required to organize the mature neuromuscular synapse by regulating synaptic vesicle availability.

The necroptosis machinery mediates axonal degeneration in a model of Parkinson disease.

Correction: The necroptosis machinery mediates axonal degeneration in a model of Parkinson disease.

The necroptosis pathway and its role in age-related neurodegenerative diseases: will it open up new therapeutic avenues in the next decade?

Schwann cell reprogramming into repair cells increases miRNA-21 expression in exosomes promoting axonal growth.

Ex Vivo Analysis of Axonal Degeneration Using Sciatic and Optic Nerve Preparations.

Collateral Sprouting of Peripheral Sensory Neurons Exhibits a Unique Transcriptomic Profile.

Axonal Degeneration in AD: The Contribution of Aβ and Tau.

GERO Cohort Protocol, Chile, 2017-2022: Community-based Cohort of Functional Decline in Subjective Cognitive Complaint elderly.

New insights on the molecular mechanisms of collateral sprouting after peripheral nerve injury.

Dementia in Latin America: Paving the way toward a regional action plan.

Author Correction: IRE1α governs cytoskeleton remodeling and cell migration through a direct interaction with filamin A.

Enhanced Activity of Exportin-1/CRM1 in Neurons Contributes to Autophagy Dysfunction and Senescent Features in Old Mouse Brain.

Aβ oligomers trigger necroptosis-mediated neurodegeneration via microglia activation in Alzheimer's disease.

Commentary on "PANoptosis-like cell death in ischemia/reperfusion injury of retinal neurons".