Katia Cailliau

The Yersinia pseudotuberculosis Yut protein, a new type of urea transporter homologous to eukaryotic channels and functionally interchangeable in vitro with the Helicobacter pylori UreI protein.

The adapter protein ZIP binds Grb14 and regulates its inhibitory action on insulin signaling by recruiting protein kinase Czeta.

Inhibition of FGF receptor signalling in Xenopus oocytes: differential effect of Grb7, Grb10 and Grb14.

The PIR domain of Grb14 is an intrinsically unstructured protein: implication in insulin signaling.

Conservation of epidermal growth factor receptor function in the human parasitic helminth Schistosoma mansoni.

ERK2 is required for FGF1-induced JNK1 phosphorylation in Xenopus oocyte expressing FGF receptor 1.

The Ste20-like kinase SLK is required for cell cycle progression through G2.

FGF receptor phosphotyrosine 766 is a target for Grb14 to inhibit MDA-MB-231 human breast cancer cell signaling.

Characterization of Schistosoma mansoni Sds homologue, a leucine-rich repeat protein that interacts with protein phosphatase type 1 and interrupts a G2/M cell-cycle checkpoint.

Regulation of protein phosphatase type 1 and cell cycle progression by PfLRR1, a novel leucine-rich repeat protein of the human malaria parasite Plasmodium falciparum.

The alphavirus 6K protein activates endogenous ionic conductances when expressed in Xenopus oocytes.

Molecular cloning and characterisation of SmSLK, a novel Ste20-like kinase in Schistosoma mansoni.

A Toxoplasma gondii leucine-rich repeat protein binds phosphatase type 1 protein and negatively regulates its activity.

A non-canonical Grb2-PLC-gamma1-Sos cascade triggered by lipovitellin 1, an apolipoprotein B homologue.

A microinjectable biological system, the Xenopus oocyte, as an approach to understanding signal transduction protein function.

Molecular determinants of Grb14-mediated inhibition of insulin signaling.

Akt interaction with PLC(gamma) regulates the G(2)/M transition triggered by FGF receptors from MDA-MB-231 breast cancer cells.

Schistosoma mansoni Polo-like kinase 1: A mitotic kinase with key functions in parasite reproduction.

Calcium dynamics during physiological acidification in Xenopus oocyte.

Grb14 inhibits FGF receptor signaling through the regulation of PLCγ recruitment and activation.

Schistosoma mansoni: structural and biochemical characterization of two distinct Venus Kinase Receptors.

Characterization of the Src/Abl hybrid kinase SmTK6 of Schistosoma mansoni.

SmSak, the second Polo-like kinase of the helminth parasite Schistosoma mansoni: conserved and unexpected roles in meiosis.

Discovery of platyhelminth-specific α/β-integrin families and evidence for their role in reproduction in Schistosoma mansoni.

Dual targeting of insulin and venus kinase Receptors of Schistosoma mansoni for novel anti-schistosome therapy.

Transcriptome Analyses of Inhibitor-treated Schistosome Females Provide Evidence for Cooperating Src-kinase and TGFβ Receptor Pathways Controlling Mitosis and Eggshell Formation.

PhosphoTyrosyl phosphatase activator of Plasmodium falciparum: identification of its residues involved in binding to and activation of PP2A.

Venus kinase receptors control reproduction in the platyhelminth parasite Schistosoma mansoni.

Targeting Echinococcus multilocularis stem cells by inhibition of the Polo-like kinase EmPlk1.

Imatinib treatment causes substantial transcriptional changes in adult Schistosoma mansoni in vitro exhibiting pleiotropic effects.

Gonad RNA-specific qRT-PCR analyses identify genes with potential functions in schistosome reproduction such as SmFz1 and SmFGFRs.

Compound library screening identified Akt/PKB kinase pathway inhibitors as potential key molecules for the development of new chemotherapeutics against schistosomiasis.

Insulin-induced cell division is controlled by the adaptor Grb14 in a Chfr-dependent manner.

Nitric Oxide Donor s-Nitroso-n-Acetyl Penicillamine (SNAP) Alters Meiotic Spindle Morphogenesis in Xenopus Oocytes.

RasGAP Shields Akt from Deactivating Phosphatases in Fibroblast Growth Factor Signaling but Loses This Ability Once Cleaved by Caspase-3.