Vladislav Levchenko

Histone release during transcription: NAP1 forms a complex with H2A and H2B and facilitates a topologically dependent release of H3 and H4 from the nucleosome.

Sterol carrier protein-2-facilitated intermembrane transfer of cholesterol- and phospholipid-derived hydroperoxides.

Histone release during transcription: displacement of the two H2A-H2B dimers in the nucleosome is dependent on different levels of transcription-induced positive stress.

Intracellular dissemination of peroxidative stress. Internalization, transport, and lethal targeting of a cholesterol hydroperoxide species by sterol carrier protein-2-overexpressing hepatoma cells.

Novel enrichment of tumor cell transfectants expressing high levels of type 4 glutathione peroxidase using 7alpha-hydroperoxycholesterol as a selection agent.

Regulation of ENaC expression at the cell surface by Rab11.

Peroxisome proliferator-activated receptor gamma antagonists decrease Na+ transport via the epithelial Na+ channel.

EGF and its related growth factors mediate sodium transport in mpkCCDc14 cells via ErbB2 (neu/HER-2) receptor.

Endothelin-1 inhibits the epithelial Na+ channel through betaPix/14-3-3/Nedd4-2.

Novel role of Rac1/WAVE signaling mechanism in regulation of the epithelial Na+ channel.

NSAIDs acutely inhibit TRPC channels in freshly isolated rat glomeruli.

Cortical actin binding protein cortactin mediates ENaC activity via Arp2/3 complex.

Effects of cytochrome P-450 metabolites of arachidonic acid on the epithelial sodium channel (ENaC).

ROS production as a common mechanism of ENaC regulation by EGF, insulin, and IGF-1.

Regulation of ENaC in mice lacking renal insulin receptors in the collecting duct.

Real-time electrochemical detection of ATP and H₂O₂ release in freshly isolated kidneys.

Deficiency of renal cortical EGF increases ENaC activity and contributes to salt-sensitive hypertension.

Pharmacological characterization of the P2 receptors profile in the podocytes of the freshly isolated rat glomeruli.

Role of Rho GDP dissociation inhibitor α in control of epithelial sodium channel (ENaC)-mediated sodium reabsorption.

Impaired epithelial Na+ channel activity contributes to cystogenesis and development of autosomal recessive polycystic kidney disease in PCK rats.

Cross-talk between insulin and IGF-1 receptors in the cortical collecting duct principal cells: implication for ENaC-mediated Na+ reabsorption.