Francesco Papaleo

The corticotropin-releasing factor receptor-1 pathway mediates the negative affective states of opiate withdrawal.

Gender- and morphine dose-linked expression of spontaneous somatic opiate withdrawal in mice.

Disruption of the CRF/CRF1 receptor stress system exacerbates the somatic signs of opiate withdrawal.

Decreased motivation to eat in mu-opioid receptor-deficient mice.

Disruption of the CRF(2) receptor pathway decreases the somatic expression of opiate withdrawal.

Genetic dissection of the role of catechol-O-methyltransferase in cognition and stress reactivity in mice.

Role of dysbindin in dopamine receptor trafficking and cortical GABA function.

Dysbindin and Schizophrenia: it's dopamine and glutamate all over again.

Mouse models of genetic effects on cognition: relevance to schizophrenia.

Working memory deficits, increased anxiety-like traits, and seizure susceptibility in BDNF overexpressing mice.

Editorial: COMT as a drug target for nervous system disorders.

COMT implication in cognitive and psychiatric symptoms in chromosome 22q11 microdeletion syndrome: a selective review.

COMT as a drug target for cognitive functions and dysfunctions.

Neuregulin 1-ErbB4-PI3K signaling in schizophrenia and phosphoinositide 3-kinase-p110δ inhibition as a potential therapeutic strategy.

[Subtypes of psychotic-like experiences in a community sample of young adults: socio-demographic correlates and substance use].

Effects of sex and COMT genotype on environmentally modulated cognitive control in mice.

Loss of dysbindin-1 in mice impairs reward-based operant learning by increasing impulsive and compulsive behavior.

The ultimate intra-/extra-dimensional attentional set-shifting task for mice.

Automatic visual tracking and social behaviour analysis with multiple mice.

Chronic and acute intranasal oxytocin produce divergent social effects in mice.

COMT Genetic Reduction Produces Sexually Divergent Effects on Cortical Anatomy and Working Memory in Mice and Humans.

Dopaminergic function in relation to genes associated with risk for schizophrenia: translational mutant mouse models.

Indicated prevention with long-chain polyunsaturated omega-3 fatty acids in patients with 22q11DS genetically at high risk for psychosis. Protocol of a randomized, double-blind, placebo-controlled treatment trial.

Sex-dichotomous effects of functional COMT genetic variations on cognitive functions disappear after menopause in both health and schizophrenia.