Peter Ferrazzano

The brain in the age of old: the hippocampal formation is targeted differentially by diseases of late life.

Inhibition of Na+/H+ exchanger isoform 1 is neuroprotective in neonatal hypoxic ischemic brain injury.

Excessive Na+/H+ exchange in disruption of dendritic Na+ and Ca2+ homeostasis and mitochondrial dysfunction following in vitro ischemia.

Activation of microglia depends on Na+/H+ exchange-mediated H+ homeostasis.

Inhibiting the Na+/H+ exchanger reduces reperfusion injury: a small animal MRI study.

Chronic neurological deficits in mice after perinatal hypoxia and ischemia correlate with hemispheric tissue loss and white matter injury detected by MRI.

Role of sodium/hydrogen exchanger isoform 1 in microglial activation and proinflammatory responses in ischemic brains.

Diffusion tensor MRI of spinal decompression sickness.

TrkB receptor agonist 7, 8 dihydroxyflavone triggers profound gender- dependent neuroprotection in mice after perinatal hypoxia and ischemia.

Age-dependent microglial activation in immature brains after hypoxia- ischemia.

Stimulation of Na(+)/H(+) exchanger isoform 1 promotes microglial migration.

Sustained Na+/H+ exchanger activation promotes gliotransmitter release from reactive hippocampal astrocytes following oxygen-glucose deprivation.

Correlation of EEG with neuropsychological status in children with epilepsy.

ERα Signaling Is Required for TrkB-Mediated Hippocampal Neuroprotection in Female Neonatal Mice after Hypoxic Ischemic Encephalopathy(1,2,3).

Suppression of microglia activation after hypoxia-ischemia results in age-dependent improvements in neurologic injury.

Monitoring Ischemic Cerebral Injury in Spontaneously Hypertensive Rats by Diffusion Tensor Imaging.