Andrea M. Vincent

Oxidative stress and programmed cell death in diabetic neuropathy.

Control of cell survival by IGF signaling pathways.

High glucose-induced oxidative stress and mitochondrial dysfunction in neurons.

Identification of the neuroprotective molecular region of pigment epithelium-derived factor and its binding sites on motor neurons.

The role of growth factors in diabetic peripheral neuropathy.

Uncoupling proteins prevent glucose-induced neuronal oxidative stress and programmed cell death.

Neuroinflammation, COX-2, and ALS--a dual role?

IGF-I prevents glutamate-induced motor neuron programmed cell death.

New insights into the mechanisms of diabetic neuropathy.

Oxidative stress in the pathogenesis of diabetic neuropathy.

Induction of antioxidant enzymes in murine podocytes precedes injury by puromycin aminonucleoside.

Short-term hyperglycemia produces oxidative damage and apoptosis in neurons.

Adeno-associated viral-mediated insulin-like growth factor delivery protects motor neurons in vitro.

Cell culture modeling to test therapies against hyperglycemia-mediated oxidative stress and injury.

Receptor for advanced glycation end products activation injures primary sensory neurons via oxidative stress.

SOD2 protects neurons from injury in cell culture and animal models of diabetic neuropathy.

Can drug screening lead to candidate therapies for testing in diabetic neuropathy?

Strategic approaches to developing drug treatments for ALS.

The antioxidant response as a drug target in diabetic neuropathy.

Oxidative injury and neuropathy in diabetes and impaired glucose tolerance.

Diabetic neuropathy: mechanisms to management.

Sensory neurons and schwann cells respond to oxidative stress by increasing antioxidant defense mechanisms.

Dyslipidemia-induced neuropathy in mice: the role of oxLDL/LOX-1.

Hyperlipidemia: a new therapeutic target for diabetic neuropathy.

Mitochondrial biogenesis and fission in axons in cell culture and animal models of diabetic neuropathy.

Diabetic neuropathy: cellular mechanisms as therapeutic targets.