Laura S. Haneline

Fanconi anemia type C-deficient hematopoietic stem/progenitor cells exhibit aberrant cell cycle control.

Distinct pathways involving the FK506-binding proteins 12 and 12.6 underlie IL-2-versus IL-15-mediated proliferation of T cells.

Oxidant hypersensitivity of Fanconi anemia type C-deficient cells is dependent on a redox-regulated apoptotic pathway.

BMP10 is essential for maintaining cardiac growth during murine cardiogenesis.

Continuous in vivo infusion of interferon-gamma (IFN-gamma) preferentially reduces myeloid progenitor numbers and enhances engraftment of syngeneic wild-type cells in Fancc-/- mice.

Ex vivo culture of Fancc-/- stem/progenitor cells predisposes cells to undergo apoptosis, and surviving stem/progenitor cells display cytogenetic abnormalities and an increased risk of malignancy.

Enhanced TNF-alpha-induced apoptosis in Fanconi anemia type C-deficient cells is dependent on apoptosis signal-regulating kinase 1.

Genetic reduction of class IA PI-3 kinase activity alters fetal hematopoiesis and competitive repopulating ability of hematopoietic stem cells in vivo.

Continuous in vivo infusion of interferon-gamma (IFN-gamma) enhances engraftment of syngeneic wild-type cells in Fanca-/- and Fancg-/- mice.

Human CD34+AC133+VEGFR-2+ cells are not endothelial progenitor cells but distinct, primitive hematopoietic progenitors.

Cul4A is required for hematopoietic stem-cell engraftment and self-renewal.

In vitro hyperglycemia or a diabetic intrauterine environment reduces neonatal endothelial colony-forming cell numbers and function.

Endothelial colony forming cells and mesenchymal stem cells are enriched at different gestational ages in human umbilical cord blood.

Oxidative stress impairs endothelial progenitor cell function.

Redox regulation of stem and progenitor cells.

Suppressed hindlimb perfusion in Rac2-/- and Nox2-/- mice does not result from impaired collateral growth.

Distinct roles of stress-activated protein kinases in Fanconi anemia-type C-deficient hematopoiesis.

Endothelial abnormalities in adolescents with type 1 diabetes: a biomarker for vascular sequelae?

Gestational diabetes mellitus alters maternal and neonatal circulating endothelial progenitor cell subsets.

Dishevelled-associated activator of morphogenesis 1 (Daam1) is required for heart morphogenesis.

Fanconi anemia D2 protein is an apoptotic target mediated by caspases.

Restrictive loss of plakoglobin in cardiomyocytes leads to arrhythmogenic cardiomyopathy.

Impaired function of Fanconi anemia type C-deficient macrophages.

Lack of plakoglobin in epidermis leads to keratoderma.

Gestational diabetes induces alterations in the function of neonatal endothelial colony-forming cells.

RESPONSE TO LETTER TO THE EDITOR - "Critical assessment of putative endothelial progenitor phenotypes" by Gian Paolo Fadini, et al.

Introduction. Common drugs in pregnancy: an update from the Obstetric-Fetal Pharmacology Research Unit Network.

Fetal hyperglycemia and a high-fat diet contribute to aberrant glucose tolerance and hematopoiesis in adult rats.

Lung parenchymal development in premature infants without bronchopulmonary dysplasia.

Epigenetic Regulation of Placenta-Specific 8 Contributes to Altered Function of Endothelial Colony-Forming Cells Exposed to Intrauterine Gestational Diabetes Mellitus.

Biomarkers for Diagnosis and Prognosis of Sinusoidal Obstruction Syndrome after Hematopoietic Cell Transplantation.

Mesenchyme Homeobox 2 Enhances Migration of Endothelial Colony Forming Cells Exposed to Intrauterine Diabetes Mellitus.

Kinetic analyses of vasculogenesis inform mechanistic studies.