Camilla Tincati

Treatment interruptions in HIV-infected subjects.

FoxP3 mRNA expression in regulatory T cells from patients with tuberculosis.

Bone diseases associated with human immunodeficiency virus infection: pathogenesis, risk factors and clinical management.

Subclinical hypothyroidism in HIV-infected subjects.

Early impairment of gut function and gut flora supporting a role for alteration of gastrointestinal mucosa in human immunodeficiency virus pathogenesis.

Untangling the immunological implications of nadir on CD4+ cell recovery during suppressive highly active antiretroviral therapy.

Insulin resistance affects early virologic response in HIV-infected subjects treated for hepatitis C infection.

The challenge of IL-2 immunotherapy in HIV disease: "no through road" or turning point?

Serum alpha-fetoprotein levels predict early virologic response in HIV-positive subjects treated for chronic hepatitis C.

Microbial translocation is associated with sustained failure in CD4+ T-cell reconstitution in HIV-infected patients on long-term highly active antiretroviral therapy.

Abacavir and cardiovascular risk in HIV-infected patients: does T lymphocyte hyperactivation exert a pathogenic role?

Sudden cardiac death in a young HIV-positive man on effective antiretroviral therapy.

The absence of CD4+ T cell count recovery despite receipt of virologically suppressive highly active antiretroviral therapy: clinical risk, immunological gaps, and therapeutic options.

Early initiation of highly active antiretroviral therapy fails to reverse immunovirological abnormalities in gut-associated lymphoid tissue induced by acute HIV infection.

CD8+ hyperactivation and senescence correlate with early carotid intima-media thickness in HIV+ patients with no cardiovascular disease.

IL-2 immunotherapy to recently HIV-1 infected adults maintains the numbers of IL-17 expressing CD4+ T (T(H)17) cells in the periphery.

Reduced CD127 expression on peripheral CD4+ T cells impairs immunological recovery in course of suppressive highly active antiretroviral therapy.

A comprehensive ex vivo functional analysis of human NKT cells reveals production of MIP1-α and MIP1-β, a lack of IL-17, and a Th1-bias in males.

Evidence for polymicrobic flora translocating in peripheral blood of HIV-infected patients with poor immune response to antiretroviral therapy.

Reduced Central Memory CD4+ T Cells and Increased T-Cell Activation Characterise Treatment-Naive Patients Newly Diagnosed at Late Stage of HIV Infection.