Edward H. Bertram

Design and construction of a long-term continuous video-EEG monitoring unit for simultaneous recording of multiple small animals.

Functional anatomy of limbic epilepsy: a proposal for central synchronization of a diffusely hyperexcitable network.

The pathological substrate of limbic epilepsy: neuronal loss in the medial dorsal thalamic nucleus as the consistent change.

Ketamine controls prolonged status epilepticus.

The midline thalamus: alterations and a potential role in limbic epilepsy.

Diminished allopregnanolone enhancement of GABA(A) receptor currents in a rat model of chronic temporal lobe epilepsy.

Phenobarbital and MK-801, but not phenytoin, improve the long-term outcome of status epilepticus.

Interictal and postictal alterations of pulsatile secretions of luteinizing hormone in temporal lobe epilepsy in men.

Depression in intractable partial epilepsy varies by laterality of focus and surgery.

Why does surgery fail to cure limbic epilepsy? Seizure functional anatomy may hold the answer.

A comparison of three NMDA receptor antagonists in the treatment of prolonged status epilepticus.

In situ-produced 7-chlorokynurenate has different effects on evoked responses in rats with limbic epilepsy in comparison to naive controls.

Interictal and postictal circadian and ultradian luteinizing hormone secretion in men with temporal lobe epilepsy.

Selective loss of dentate hilar interneurons contributes to reduced synaptic inhibition of granule cells in an electrical stimulation-based animal model of temporal lobe epilepsy.

The relevance of kindling for human epilepsy.

Alterations in GABA(A) receptor mediated inhibition in adjacent dorsal midline thalamic nuclei in a rat model of chronic limbic epilepsy.

Multiple roles of midline dorsal thalamic nuclei in induction and spread of limbic seizures.

Changes in midline thalamic recruiting responses in the prefrontal cortex of the rat during the development of chronic limbic seizures.

Temporal lobe epilepsy: where do the seizures really begin?

Modulation of sodium channel inactivation gating by a novel lactam: implications for seizure suppression in chronic limbic epilepsy.

Altered pharmacology and GABA-A receptor subunit expression in dorsal midline thalamic neurons in limbic epilepsy.

Workshop on idiopathic generalized epilepsies: Bridging basic science and clinical research (October 3-6, 2007; Antalya, Turkey).

Adenosine A1 receptors presynaptically modulate excitatory synaptic input onto subiculum neurons.

Counterpoint to "what is an epileptic seizure?" by D'Ambrosio and Miller.

Temporal lobe epilepsy induces intrinsic alterations in Na channel gating in layer II medial entorhinal cortex neurons.

Mesial temporal lobe epilepsy: How do we improve surgical outcome?

Increased GABAergic inhibition in the midline thalamus affects signaling and seizure spread in the hippocampus-prefrontal cortex pathway.

Excitatory amplification through divergent-convergent circuits: the role of the midline thalamus in limbic seizures.

[Why don't medicines for epilepsy always work?].

Enhanced actions of adenosine in medial entorhinal cortex layer II stellate neurons in temporal lobe epilepsy are mediated via A(1)-receptor activation.

Neuronal circuits in epilepsy: do they matter?

Evidence for a role of Nav1.6 in facilitating increases in neuronal hyperexcitability during epileptogenesis.

Epilepsy biomarkers.

Electrophysiology in epilepsy surgery: Roles and limitations.

Extratemporal lobe circuits in temporal lobe epilepsy.

A model of posttraumatic epilepsy after penetrating brain injuries: effect of lesion size and metal fragments.

Suppressing limbic seizures by stimulating medial dorsal thalamic nucleus: factors for efficacy.

Non-Invasive, Focal Disconnection of Brain Circuitry Using Magnetic Resonance-Guided Low-Intensity Focused Ultrasound to Deliver a Neurotoxin.

Genetic resistance to kindling associated with alterations in circuit function.

Modelling traumatic brain injury and posttraumatic epilepsy in rodents.

Testing Different Combinations of Acoustic Pressure and Doses of Quinolinic Acid for Induction of Focal Neuron Loss in Mice Using Transcranial Low-Intensity Focused Ultrasound.

Inhibition of T-Type calcium channels in mEC layer II stellate neurons reduces neuronal hyperexcitability associated with epilepsy.

Effects of Non-invasive, Targeted, Neuronal Lesions on Seizures in a Mouse Model of Temporal Lobe Epilepsy.