Eliza Wiercinska

Smad7 prevents activation of hepatic stellate cells and liver fibrosis in rats.

Transdifferentiation-dependent expression of alpha-SMA in hepatic stellate cells does not involve TGF-beta pathways leading to coinduction of collagen type I and thrombospondin-2.

Anti-TGF-beta strategies for the treatment of chronic liver disease.

Id1 is a critical mediator in TGF-beta-induced transdifferentiation of rat hepatic stellate cells.

Liver fibrogenesis due to cholestasis is associated with increased Smad7 expression and Smad3 signaling.

Transforming growth factor beta1 up-regulates interferon regulatory factor 8 during dendritic cell development.

The bone morphogenetic protein pathway is inactivated in the majority of sporadic colorectal cancers.

5-Aminosalicylic acid inhibits TGF-beta1 signalling in colorectal cancer cells.

Transforming growth factor-beta induces nerve growth factor expression in pancreatic stellate cells by activation of the ALK-5 pathway.

TMEPAI, a transmembrane TGF-beta-inducible protein, sequesters Smad proteins from active participation in TGF-beta signaling.

Matrix metalloproteinase-14 (MT1-MMP)-mediated endoglin shedding inhibits tumor angiogenesis.

The TGF-β/Smad pathway induces breast cancer cell invasion through the up-regulation of matrix metalloproteinase 2 and 9 in a spheroid invasion model system.

Oncogenic functions of hMDMX in in vitro transformation of primary human fibroblasts and embryonic retinoblasts.

BMP-7 inhibits TGF-β-induced invasion of breast cancer cells through inhibition of integrin β(3) expression.