Courtney M. Karner

Molecular regulation of kidney development: is the answer blowing in the Wnt?

Mutations of HNF-1beta inhibit epithelial morphogenesis through dysregulation of SOCS-3.

Wnt9b signaling regulates planar cell polarity and kidney tubule morphogenesis.

Aberrant planar cell polarity induced by urinary tract obstruction.

Lrp4 regulates initiation of ureteric budding and is crucial for kidney formation--a mouse model for Cenani-Lenz syndrome.

Tankyrase is necessary for canonical Wnt signaling during kidney development.

Integrin-linked kinase regulates p38 MAPK-dependent cell cycle arrest in ureteric bud development.

Canonical Wnt9b signaling balances progenitor cell expansion and differentiation during kidney development.

Diverse chemical scaffolds support direct inhibition of the membrane-bound O-acyltransferase porcupine.

Vertebrate kidney tubules elongate using a planar cell polarity-dependent, rosette-based mechanism of convergent extension.

Stromal-epithelial crosstalk regulates kidney progenitor cell differentiation.

Wnt4 is essential to normal mammalian lung development.

Biphasic regulation of glutamine consumption by WNT during osteoblast differentiation.

Obesity alters the collagen organization and mechanical properties of murine cartilage.

An adhesion G protein-coupled receptor is required in cartilaginous and dense connective tissues to maintain spine alignment.

SLC1A5 provides glutamine and asparagine necessary for bone development in mice.

Bioenergetic Metabolism In Osteoblast Differentiation.

Distinct Roles of Glutamine Metabolism in Benign and Malignant Cartilage Tumors With IDH Mutations.

SLC38A2 provides proline to fulfil unique synthetic demands arising during osteoblast differentiation and bone formation.