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The quiescent fraction of chronic myeloid leukemic stem cells depends on BMPR1B, Stat3 and BMP4-niche signals to persist in patients in remission.
Haematologica Jan, 2021 | Pubmed ID: 32001529
Primitive CML cell expansion relies on abnormal levels of BMPs provided by the niche and on BMPRIb overexpression.
Blood Nov, 2013 | Pubmed ID: 24100446
Immature CML cells implement a BMP autocrine loop to escape TKI treatment.
Blood Dec, 2017 | Pubmed ID: 29138221
A new signaling cascade linking BMP4, BMPR1A, ΔNp73 and NANOG impacts on stem-like human cell properties and patient outcome.
Cell death & disease Sep, 2018 | Pubmed ID: 30262802
Antimetabolic cooperativity with the clinically approved l-asparaginase and tyrosine kinase inhibitors to eradicate CML stem cells.
Molecular metabolism Jan, 2022 | Pubmed ID: 34863941
A minimal standardized human bone marrow microphysiological system to assess resident cell behavior during normal and pathological processes.
Biomaterials science Jan, 2022 | Pubmed ID: 34904143
Centre de Recherche en Cancérologie de Lyon
Université de Lyon
Centre Léon Bérard
Kawtar Arizkane1,2,3,
Kevin Geistlich1,2,3,4,
Laurine Moindrot1,2,3,
Emma Risson1,2,3,
Sandrine Jeanpierre1,2,3,4,
Léa Barral1,2,3,
Anaëlle Bobard1,2,3,
Giulio Menegazzi5,
Thibault Voeltzel1,2,3,
Véronique Maguer-Satta1,2,3,
Sylvain Lefort1,2,3
1, Centre de Recherche en Cancérologie de Lyon,
2, Centre de Recherche en Cancérologie de Lyon,
3, Université de Lyon,
4, Centre Léon Bérard,
5Department of Experimental and Clinical Biomedical Sciences Mario Serio, Università degli Studi di Firenze
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