George Perides

Biliary acute pancreatitis in mice is mediated by the G-protein-coupled cell surface bile acid receptor Gpbar1.

Role of aggrecanase 1 in Lyme arthritis.

Cause-effect relationships between zymogen activation and other early events in secretagogue-induced acute pancreatitis.

Tumor progression locus-2 is a critical regulator of pancreatic and lung inflammation during acute pancreatitis.

A mouse model of acute biliary pancreatitis induced by retrograde pancreatic duct infusion of Na-taurocholate.

'Role reversal' for the receptor PAR1 in sepsis-induced vascular damage.

Protease-activated receptor-2 exerts contrasting model-specific effects on acute experimental pancreatitis.

Matrix metalloproteinase 9 plays a key role in lyme arthritis but not in dissemination of Borrelia burgdorferi.

Experimental acute biliary pancreatitis induced by retrograde infusion of bile acids into the mouse pancreatic duct.

TNF-alpha-dependent regulation of acute pancreatitis severity by Ly-6C(hi) monocytes in mice.

Interdicting protease-activated receptor-2-driven inflammation with cell-penetrating pepducins.

Ryanodine receptors contribute to bile acid-induced pathological calcium signaling and pancreatitis in mice.

Bile acids induce pancreatic acinar cell injury and pancreatitis by activating calcineurin.

Mechanically Unloading the Left Ventricle Before Coronary Reperfusion Reduces Left Ventricular Wall Stress and Myocardial Infarct Size.