Yiorgos Apidianakis

Challenge of Drosophila melanogaster with Cryptococcus neoformans and role of the innate immune response.

Profiling early infection responses: Pseudomonas aeruginosa eludes host defenses by suppressing antimicrobial peptide gene expression.

Hedgehog restricts its expression domain in the Drosophila wing.

Involvement of skeletal muscle gene regulatory network in susceptibility to wound infection following trauma.

Drosophila melanogaster as a model host for studying Pseudomonas aeruginosa infection.

The pathogenic properties of a novel and conserved gene product, KerV, in proteobacteria.

Homeostasis in infected epithelia: stem cells take the lead.

Synergy between bacterial infection and genetic predisposition in intestinal dysplasia.

Homeostatic interplay between bacterial cell-cell signaling and iron in virulence.

In vivo high-resolution magic angle spinning magnetic resonance spectroscopy of Drosophila melanogaster at 14.1 T shows trauma in aging and in innate immune-deficiency is linked to reduced insulin signaling.

Drosophila melanogaster as a model for human intestinal infection and pathology.

A quorum sensing regulated small volatile molecule reduces acute virulence and promotes chronic infection phenotypes.

Down-regulation of glutatione S-transferase α 4 (hGSTA4) in the muscle of thermally injured patients is indicative of susceptibility to bacterial infection.