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Challenge of Drosophila melanogaster with Cryptococcus neoformans and role of the innate immune response.
Eukaryotic cell Apr, 2004 | Pubmed ID: 15075271
Profiling early infection responses: Pseudomonas aeruginosa eludes host defenses by suppressing antimicrobial peptide gene expression.
Proceedings of the National Academy of Sciences of the United States of America Feb, 2005 | Pubmed ID: 15695583
Hedgehog restricts its expression domain in the Drosophila wing.
EMBO reports Aug, 2007 | Pubmed ID: 17571073
Involvement of skeletal muscle gene regulatory network in susceptibility to wound infection following trauma.
PloS one , 2007 | Pubmed ID: 18159239
Drosophila melanogaster as a model host for studying Pseudomonas aeruginosa infection.
Nature protocols , 2009 | Pubmed ID: 19680242
The pathogenic properties of a novel and conserved gene product, KerV, in proteobacteria.
PloS one , 2009 | Pubmed ID: 19779606
Homeostasis in infected epithelia: stem cells take the lead.
Cell host & microbe Oct, 2009 | Pubmed ID: 19837370
Synergy between bacterial infection and genetic predisposition in intestinal dysplasia.
Proceedings of the National Academy of Sciences of the United States of America Dec, 2009 | Pubmed ID: 19934041
Homeostatic interplay between bacterial cell-cell signaling and iron in virulence.
PLoS pathogens Mar, 2010 | Pubmed ID: 20300606
In vivo high-resolution magic angle spinning magnetic resonance spectroscopy of Drosophila melanogaster at 14.1 T shows trauma in aging and in innate immune-deficiency is linked to reduced insulin signaling.
International journal of molecular medicine Aug, 2010 | Pubmed ID: 20596596
Drosophila melanogaster as a model for human intestinal infection and pathology.
Disease models & mechanisms Jan, 2011 | Pubmed ID: 21183483
A quorum sensing regulated small volatile molecule reduces acute virulence and promotes chronic infection phenotypes.
PLoS pathogens Aug, 2011 | Pubmed ID: 21829370
Down-regulation of glutatione S-transferase α 4 (hGSTA4) in the muscle of thermally injured patients is indicative of susceptibility to bacterial infection.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology Feb, 2012 | Pubmed ID: 22038048
Shriners Burn Institute
Massachusetts General Hospital, Harvard Medical School
Valeria Righi1,2,3,
Yiorgos Apidianakis2,4,
Laurence G. Rahme2,4,
A. Aria Tzika1,2,3
1NMR Surgical Laboratory, Department of Surgery, Massachusetts General Hospital, Harvard Medical School,
2, Shriners Burn Institute,
3Department of Radiology, Athinoula A. Martinos Center of Biomedical Imaging, Harvard Medical School,
4Molecular Surgery Laboratory, Department of Surgery, Massachusetts General Hospital, Harvard Medical School
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