Stephanie S. Watowich

Oncogene cooperativity in Friend erythroleukemia: erythropoietin receptor activation by the env gene of SFFV leads to transcriptional upregulation of PU.1, independent of SFFV proviral insertion.

Control of myeloid-specific integrin alpha Mbeta 2 (CD11b/CD18) expression by cytokines is regulated by Stat3-dependent activation of PU.1.

Differential regulation of SOCS genes in normal and transformed erythroid cells.

Hematopoietic cell survival signals are elicited through non-tyrosine-containing sequences in the membrane-proximal region of the erythropoietin receptor (EPOR) by a Stat5-dependent pathway.

Enhancer-mediated control of macrophage-specific arginase I expression.

Cytokine signals through STAT3 promote expression of granulocyte secondary granule proteins in 32D cells.

HIF-1alpha, STAT3, CBP/p300 and Ref-1/APE are components of a transcriptional complex that regulates Src-dependent hypoxia-induced expression of VEGF in pancreatic and prostate carcinomas.

IL-10-independent STAT3 activation by Toxoplasma gondii mediates suppression of IL-12 and TNF-alpha in host macrophages.

A Ras homologue member I directly inhibits signal transducers and activators of transcription 3 translocation and activity in human breast and ovarian cancer cells.

IL-6 signaling via the STAT3/SOCS3 pathway: functional analysis of the conserved STAT3 N-domain.

Src activation of Stat3 is an independent requirement from NF-kappaB activation for constitutive IL-8 expression in human pancreatic adenocarcinoma cells.

STAT3 governs distinct pathways in emergency granulopoiesis and mature neutrophils.

General nature of the STAT3-activated anti-inflammatory response.

STAT3 regulates cytokine-mediated generation of inflammatory helper T cells.

Mutations in the cofilin partner Aip1/Wdr1 cause autoinflammatory disease and macrothrombocytopenia.

Essential autocrine regulation by IL-21 in the generation of inflammatory T cells.

Cutting edge: A transcriptional repressor and corepressor induced by the STAT3-regulated anti-inflammatory signaling pathway.

T helper 17 lineage differentiation is programmed by orphan nuclear receptors ROR alpha and ROR gamma.

The signal transducer STAT5 inhibits plasmacytoid dendritic cell development by suppressing transcription factor IRF8.

Granulocyte colony-stimulating factor: molecular mechanisms of action during steady state and 'emergency' hematopoiesis.

Molecular antagonism and plasticity of regulatory and inflammatory T cell programs.

Generation of T follicular helper cells is mediated by interleukin-21 but independent of T helper 1, 2, or 17 cell lineages.

Dendritic cells: transcriptional control of plasmacytoid dendritic cell development by E2-2.

CCR6 regulates the migration of inflammatory and regulatory T cells.

Endogenous suppression of mast cell development and survival by IL-4 and IL-10.

Critical regulation of early Th17 cell differentiation by interleukin-1 signaling.

STAT3 controls the neutrophil migratory response to CXCR2 ligands by direct activation of G-CSF-induced CXCR2 expression and via modulation of CXCR2 signal transduction.

STAT3 controls myeloid progenitor growth during emergency granulopoiesis.

Arginine usage in mycobacteria-infected macrophages depends on autocrine-paracrine cytokine signaling.

Mechanisms regulating dendritic cell specification and development.

The erythropoietin receptor: molecular structure and hematopoietic signaling pathways.

Cell-intrinsic role for IFN-α-STAT1 signals in regulating murine Peyer patch plasmacytoid dendritic cells and conditioning an inflammatory response.

The transcriptional regulators Id2 and Id3 control the formation of distinct memory CD8+ T cell subsets.

STAT5 protein negatively regulates T follicular helper (Tfh) cell generation and function.

A STATus report on DC development.

G-CSF-activated STAT3 enhances production of the chemokine MIP-2 in bone marrow neutrophils.

The signal transducers STAT5 and STAT3 control expression of Id2 and E2-2 during dendritic cell development.

BRAF inhibition increases tumor infiltration by T cells and enhances the antitumor activity of adoptive immunotherapy in mice.

miR-22 controls Irf8 mRNA abundance and murine dendritic cell development.

The signaling suppressor CIS controls proallergic T cell development and allergic airway inflammation.

Diversification of dendritic cell subsets: Emerging roles for STAT proteins.