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Center for Thrombosis and Hemostasis Mainz,
Center for Cardiology
Sabine Kossmann has not added Biography.
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Lysozyme M-positive monocytes mediate angiotensin II-induced arterial hypertension and vascular dysfunction.
Circulation Sep, 2011 | Pubmed ID: 21875910
Inflammatory monocytes determine endothelial nitric-oxide synthase uncoupling and nitro-oxidative stress induced by angiotensin II.
The Journal of biological chemistry Oct, 2014 | Pubmed ID: 25143378
Heme oxygenase-1 suppresses a pro-inflammatory phenotype in monocytes and determines endothelial function and arterial hypertension in mice and humans.
European heart journal Dec, 2015 | Pubmed ID: 26516175
Gut Microbiota Promote Angiotensin II-Induced Arterial Hypertension and Vascular Dysfunction.
Journal of the American Heart Association Aug, 2016 | Pubmed ID: 27577581
NOX2 amplifies acetaldehyde-mediated cardiomyocyte mitochondrial dysfunction in alcoholic cardiomyopathy.
Scientific reports Sep, 2016 | Pubmed ID: 27624556
Assessment of Vascular Dysfunction and Inflammation Induced by Angiotensin II in Mice.
Methods in molecular biology (Clifton, N.J.) , 2017 | Pubmed ID: 28063062
Platelet-localized FXI promotes a vascular coagulation-inflammatory circuit in arterial hypertension.
Science translational medicine Feb, 2017 | Pubmed ID: 28148841
Distinct Regulatory Effects of Myeloid Cell and Endothelial Cell NAPDH Oxidase 2 on Blood Pressure.
Circulation May, 2017 | Pubmed ID: 28298457
University Medical Center Mainz
Jeremy Lagrange1,
Sabine Kossmann1,2,
Klytaimnistra Kiouptsi1,
Philip Wenzel1,2,3
1Center for Thrombosis and Hemostasis Mainz, University Medical Center Mainz,
2Center for Cardiology, University Medical Center Mainz,
3, German Center for Cardiovascular Research (DZHK)
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