Peter Penzes

Rapid induction of dendritic spine morphogenesis by trans-synaptic ephrinB-EphB receptor activation of the Rho-GEF kalirin.

Activity-dependent dendritic spine structural plasticity is regulated by small GTPase Rap1 and its target AF-6.

N-cadherin regulates cytoskeletally associated IQGAP1/ERK signaling and memory formation.

Kalirin-7 controls activity-dependent structural and functional plasticity of dendritic spines.

Pumping up the synapse.

Coordination of synaptic adhesion with dendritic spine remodeling by AF-6 and kalirin-7.

Dendritic spine dynamics--a key role for kalirin-7.

Convergent CaMK and RacGEF signals control dendritic structure and function.

Rapid enhancement of two-step wiring plasticity by estrogen and NMDA receptor activity.

Not just actin? A role for dynamic microtubules in dendritic spines.

Segregated populations of hippocampal principal CA1 neurons mediating conditioning and extinction of contextual fear.

Downregulation of connective tissue growth factor by three-dimensional matrix enhances ovarian carcinoma cell invasion.

Kalirin regulates cortical spine morphogenesis and disease-related behavioral phenotypes.

Epac2 induces synapse remodeling and depression and its disease-associated forms alter spines.

Kalirin loss results in cortical morphological alterations.

Myosin-Va-interacting protein, RILPL2, controls cell shape and neuronal morphogenesis via Rac signaling.

Rapid modulation of spine morphology by the 5-HT2A serotonin receptor through kalirin-7 signaling.

Disrupted-in-Schizophrenia 1 (DISC1) regulates spines of the glutamate synapse via Rac1.

Hippocampal phenotypes in kalirin-deficient mice.

Estrogen receptor ß activity modulates synaptic signaling and structure.

Epac2-mediated dendritic spine remodeling: implications for disease.

Impaired regulation of synaptic actin cytoskeleton in Alzheimer's disease.

Dendritic spine pathology in neuropsychiatric disorders.

Increased expression of Kalirin-9 in the auditory cortex of schizophrenia subjects: Its role in dendritic pathology.

Kalirin signaling: implications for synaptic pathology.

Deconstructing signal transduction pathways that regulate the actin cytoskeleton in dendritic spines.